Sleep alterations during post-traumatic coma as a possible predictor of cognitive defects.
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Biomedical subjects
Publications and source records attributed to L Rubini.
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A personal case of persistent hypoglossal artery is presented and the literature on the subject from 1967 to the present reviewed. The 16 cases collected, following on the 26 already reported in the well-known monograph of T.A. Lie on congenital anomalies of the carotid arteries, complete the statistical picture of this malformation. After discussing the embryonic origin and describing the radiological anatomy of the anomaly, the authors briefly present their case and discuss, in the light of the cases collected, the possible role of persistent hypoglossal artery in the pathogenesis of vascular diseases of the head.
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The results of surgery in 96 patients affected with cerebral abscess are reported. The lesion was completely removed in 65 patients, 33 underwent drainage (in many cases by Adso-Craig's technique) and only in one case was a combined operation performed. Overall mortality was 15.1%. More or less serious neurological and/or psychic deficits were present in 40% of patients, whereas epilepsy occurred in 26.6%. Furthermore, it was noted that excision of the abscess originates fewer epileptic complications type (20.9%) than drainage of the abscess cavity (37.5%). As regards the type of operation, the Authors suggest complete excision of the abscess as more advisable limiting drainage to cases of localization in "noble" areas or when complete removal can jeopardize patients in very serious conditions.
General remarks on carotid-ophthalmic aneurysms are followed by the description of a personal case in which direct closure of an "inoperable" aneurysm was successful. The surgical treatment of these malformations is discussed.
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The Authors report a study on recognition memory in a series of 50 severe head injuries. The effect of cerebral trauma is expressed by reduction in recognition memory and in learning. A significant difference is found between frontal lesions and temporal lesions. Left temporal lobe damage causes a more severe deficit than right temporal lobe damage. Extradural and subdural haematomas cause no such severe deficit. Neurological syndrome of level of lesion significantly correlates with the performance. PTA duration shows no relation.
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