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Biomedical subjects

M C Vagueiro

Publications and source records attributed to M C Vagueiro.

13 recordsLinked to original sources
PubMed1990

[The use of Holter electrocardiography in permanent pacemaker users].

The authors selected 37 permanent pacemaker patients followed for eleven successive months, with symptomatology eventually related to the pacing system. An Holter ambulatory monitorization was performed to all of them during 24 hours. Twenty one had single chamber systems and the others double chamber. Twelve malfunctions were found in ten patients. Six due to "undersensing", 4 related to "oversensing", and 2 from loss of capture. Two patients had tachycardia pacemaker mediated. An inadequate mode of pacing was found in other two cases. Fifteen rhythm disturbances not related to the pacemaker systems were registered in thirteen patients: supra-ventricular in 9; ventricular greater than or equal to 3 degree of lown in 5; and 2nd degree A.V.B. Mobitz type I in one case. Four patients were submitted to surgical intervention in order to modify the mode of pacing. It was also necessary to reprogram ten patients. The Holter Ambulatory Electrocardiography revealed to be an important diagnostic method for the detection of intermittent malfunction of pacing systems. The occasional changes of cardiac rhythm not linked with pacing were also revealed.

Adult↗
PubMed1978

Persistent atrial standstill with atrial inexcitability.

Electrophysiologic studies including His bundle recording, atrial, and ventricular stimulation, were performed in three symptomatic patients with persistent atrial standstill of unknown etiology. The rhythm was junctional in two cases and ventricular in one. In two cases, evidence suggestive of associated impairment of the His bundle conduction system was found. The atria were inexcitable at multiple sites and no retrograde conduction to the right atrium could be elicited by ventricular pacing. Follow-up in the three cases, respectively for 48, 42 and 12 months after pacemaker implantation, revealed no return of spontaneous atrial electrical activity.

Adult↗
PubMed1976

[Angina pectoris].

The present treatment of angina pectoris is briefly examined. It would appear from the literature published over the last three years that no cure has yet been found. Improvement in prognosis may be expected, particularly in stable forms. Existing clinical, ECG and angiocardiographic methods of diagnosis are described. Better awareness of the factors responsible for angina, coupled with more accurate diagnosis, enable clinical improvement to be achieved in a large number of cases by means of rationally planned medical or surgical management.

Angina Pectoris↗
PubMed1976

[Angina pectoris. Results of investigations to date (author's transl)].

Among the most important coronary risk factors are a diet rich in saturated fatty acids, cholesterol and calories, hypercholesterolemia, high blood pressure and excessive smoking. The typical pain can be elicited by putting a strain on the patient, e.g. climbing stairs. The disappearance of the pain after treatment with nitroglycerine is one of the most important pointers. Treatment is with nitroglycerine which, moreover, remains the drug of choice, also with nitrates such as pentaerythritol tetranitrate and with betablockers like propranolol. Favorable results have also been reported with a combination of isosorbide dinitrate and propranolol.

Adrenergic beta-Antagonists↗
PubMed1975

Electrophysiologic effects of the antiarrhythmic agent disopyramide phosphate.

The electrophysiologic effects of the antiarrhythmic agent disopyramide phosphate given intravenously were studied in 10 patients with cardiac disease. Studies included determinations of sinus recovery time and refractoriness of the atria, the atrioventricular (A-V) node and the His-Purkinje system. Measurements were performed at rest and 15 and 30 minutes after administration of disopyramide. Serum drug levels were measured at these times. Sinus recovery time was shortened at both 15 and 30 minutes, with an average decrease of 39.5 and 146.2 msec, respectively (P less than 0.01). Atrial refractoriness was not altered significantly, but tended to be reduced; the mean effective refractory period was 289.5 msec before administration of disopyramide and 259 and 270 msec 15 and 30 minutes, respectively, after administration. The functional refractory period of the atrioventricular (A-V) node was definitely prolonged in seven patients 15 minutes after administration of disopyramide. The relative refractory period of the His-Purkinje System was not altered. Although this study does not elucidate the mechanism by which disopyramide acheives its antiarrhythmic effects, animal work has shown that it is similar to that of quinidine. In the doses used the drug does not seem to cause first, second or third degree A-V block or fascicular or bundle branch block; it did not increase the severity of first degree A-V block in the three patients with this disturbance. The drug may be particularly useful when arrhythmias are associated with slow sinus rates.

Adult↗
PubMed1975

Syndrome of short P-R, narrow QRS and repetitive supraventricular tachyarrhythmias: the possible occurrence of the R-on-T phenomenon and the limits of this syndrome.

The R-on-T phenomenon was observed in two patients with narrow QRS complexes and repetitive supraventricular tachyarrhythmias. Case 1 had a typical Lown-Ganong-Levine syndrome in which the shortening of the P-R interval was due to a reduction of the A-H interval. In Case 2 the P-R interval (140 msec) was not short but the A-H interval was at the lower limits of normal (50 msec) and the P-A interval at the upper limits of normal (40 msec). Both patients showed the R-on-T phenomenon during periods of 1:1 A-V conduction occurring during atrial stimulation at a rate of 200/min (Case 1) and atrial flutter with a rate of 300/min (Case 2). Although total A-V conduction time was prolonged, the magnitude of this prolongation appeared to have been less than that occurring in normal subjects. This was attributed to the presence of an accessory pathway bypassing the area where the most significant delay occurred at the A-V node. The findings in Case 2 suggest, but do not prove, that a similar bypass might be operative in patients with atrial flutter with periods of 1:1 conduction, regardless as to whether the R-on-T phenomenon is, or is not, present.

Arrhythmias, Cardiac↗