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Biomedical subjects

M Chouza

Publications and source records attributed to M Chouza.

At least 19 recordsLinked to original sources
PubMed1990

[Multiple aneurysms of the left auricula, ascending aorta and sinuses of Valsalva with interventricular communication, fibromuscular subaortic stenosis and a single coronary artery].

We report the case of a male newborn infant with aneurysm of atrial appendage, ascending aorta and sinus of Valsalva associated to ventricular septal defect, fibromuscular subaortic stenosis and single coronary artery. The diagnosis was carried out by means of two-dimensional echocardiography and angiocardiography. This complex cardiac malformation has not been reported before.

Abnormalities, Multiple↗
PubMed1989

[Scimitar syndrome in the newborn child and infant].

We present 10 symptomatic patients with scimitar syndrome. Their age was below 1 year and four were newborn babies. In 5 patients there was secundum atrial septal defect and in one there was a patent ductus arteriosus. Sixty per cent of the patients had pulmonary sequestration. The right lung was moderately or severely hypoplasic in 8 cases and seven had frequent respiratory infections. The anomalous venous drainage produced a left-to right shunt at atrium level which in our series had a value of 2.87 +/- 0.5. Five patients had pulmonary hypertension either moderate or severe. The severity of this disease in early infancy is related to the pulmonary hypertension (40%), requiring a prompt surgical correction. Eight patients were operated upon, the drainage being connected to the left atrium. The six surviving patients are asymptomatic at 12 years of follow-up. Our series is the biggest published in our country and the second in the world literature in infancy.

Abnormalities, Multiple↗
PubMed1986

Differential diagnosis of intrapericardial mass identified as venous ectasia.

The case of an infant with an intrapericardial mass identified as venous ectasia of the left cardinal vein is presented. Because of the few cases reported in the literature of this anomaly, we believe it is of interest to update its description as an intrapericardial venous cystic dilatation, congenital in origin, and unrelated to the pericardium or the heart. Our purpose is to describe this case as a hematic cyst arising from the ectasia dilatation of the left cardinal vein as an isolated entity originating in the fetal period.

Journal Article↗
PubMed1986

[Mitral atresia. Anatomical study and surgical implications].

We present the pathological study of 17 cases of mitral atresia with patent aortic valve and their surgical implications. The study is based on the type of atrioventricular connection, presence or absence of ventricular septum, size of the left ventricle and relation of the aorta with the aforementioned ventricle. We have classified mitral atresia into two groups by assessing these data. Group A) Both ventricles and atrioventricular connections are present and there is ventricular septal defect. The aorta may connect with an either normal or hypoplastic left ventricle or it may emerge in double outlet from the right ventricle. Group B) Absent left ventricle and atrioventricular connection. The aorta emerges from the single ventricle or the rudimentary chamber. Thirteen cases belonged to group A and four to group B. The pathological structure of mitral atresia requires the decompression of the left atrium by atrioseptostomy during the neonatal period to enable survival. On a second stage a palliative correction should be carried out, which; depending on ventricular size and outlet of the vessels, will be a valvular prosthesis left atrium-left ventricle or the modified atriopulmonary technique connecting the new left atrium with the systemic ventricle and aorta after resecting the auricular septum and connecting the right atrium with the pulmonary artery.

Female↗
PubMed1984

[Univentricular heart; angiocardiographic study].

Authors present angiocardiographic study of 19 patients with univentricular heart. It was realized during the neonatal period in 13 cases and between the six first six months in the rest. To determine the existence of an only ventricle contrast was injected in the right and left atrium and the ventricle through the right and left atrioventricular valve. The bulbo-ventricular foramen was orientated in all cases from back-front wards. Those of left ventricle type had a right anterior, anterior-superior or left-anterior rudimentary chamber. Depending on its' morphology they were classified in three types: left, right or indeterminate. In these series fourteen patients had left ventricle morphology. All cases had a rudimentary chamber: 10 with transposition great vessel and four with normal related great vessels. Six presented normal situs (five with levocardia , and eight ambiguous situs (six with asplenia, four with dextrocardia). In nine atrio ventricular valves were anomalous. In the indeterminate type, two had a normal situs, two with polysplenia and two asplenia. Cardiac apex was oriented to the right in two (one asplenia). The two patients with polysplenia and one with normal situs and single atrium had a common ventricular valve. Four had an anterior aorta and three pulmonary obstruction.

Abnormalities, Multiple↗
PubMed1980

[Aberrant left pulmonary artery (author's transl)].

Two cases of aberrant left pulmonary artery are described. One of them died on the third day of life and had esophageal atresia and Fallot tetralogy as associated anomalies. Outstanding symptoms of this anomaly were bouts of respiratory difficulty with espiratory wheezing. During these episodes radiographic findings were right or bilateral lung hyperaireation and middle lobe atelectasis. Upper respiratory infections usually preceded these bouts of respiratory difficulty. Diagnosis was established by the presence of a notch on the anterior wall of the esophagus, diminished pulmonary perfusion of the left lung as shown by pulmonary gammagraphy and characteristic findings in pulmonary arteriography. During the last 12 months the first case, now aged three years, has had a spontaneous regression of symptoms.

Angiography↗
PubMed1978

[Fast atrial rhythms (author's transl)].

The authors present their experience on fast atrial rhythms. Fourteen patients were studied in the first three months of life, nine of them having congestive heart failure. The electrophysiological basis of the reentrance tachycardia at A-V junction is discussed. The pharmacological treatment is reviewed, emphasizing the usefulness of verapamil. They comment on the good prognosis of the disorder, provided there is not associated congenital heart disease.

Heart Atria↗
PubMed1977

[Corrected transposition of the great arteries. Study of 14 cases (author's transl)].

We have studied 14 patients of corrected transposition, 12 with situs solitus and 2 with situs inversus. In all the patients, excepting one, a clinical, radiological and angiocardiographical study was performed. The associated anomalies were determining elements of the clinical situation; the patients with pulmonary obstruction and interventricular communication had cianosis, while the 4 patients with aortic obstruction or anomalies type left Ebstein show cardiac insufficiency. The simple radiological study helped us to determinate the ascendent aorta in the left side of the heart, in 9 cases. It was unusual to see the droped hilium in the right hemitorax. The more significants electrical signs were: The presence of complete A-V block, Q-waves in D2, D3 and aVF, absence of right waves in precordial leads in most of the patients and a negative T wave in a VL. Summarizing, authors believe that in this type of defects selective injection of contrast in both ventricles is the best method to establish the difference between corrected transposition and other cardiopathies with the aorta situated at the left side of the pulmonary artery.

Adult↗
PubMed1975

Proximal renal tubular acidosis in the tetralogy of fallot.

A 9-year-old girl presented with tetralogy of Fallot and moderate metabolic acidosis. Despite a Blalock's fistula there was evidence of chronic hypoxia with cyanosis, clubbing of fingers and toes and very elevated blood hematocrit values. Renal acidification and bicarbonate titration demonstrated the existence of proximal renal tubular acidosis: renal bicarbonate threshold was low (18 mmoles/1) and normal urinary acidificaiton was present at subthreshold serum bicarbonate levels. Following corrective heart surgery, blood acid-base values and renal reabsorption of bicarbonate became normal. A causal relationship between extracellular fluid volume expansion dependent on the high hematocrit and proximal renal tubular acidosis is suggested.

Acidosis, Renal Tubular↗