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Biomedical subjects

M Hajzoková

Publications and source records attributed to M Hajzoková.

14 recordsLinked to original sources

Somatometric characteristic of hypermobile patients with coxarthrosis.

Different body build has been observed in hypermobile women with coxarthrosis as compared with other hypermobile individuals. The study summarizes results of somatometric measurements in 38 hypermobile women with coxarthrosis based on congenital dislocation of the hip (CDH) and the same number of hypermobile controls. The following differences were found: Patients with coxarthrosis were heavier, shorter, more robust and had a larger quantity of fat tissue. In the control group ten out of 38 (26%) showed arachnodactyly, whereas in the coxarthrosis group no arachnodactyly was found. In Heath-Carter anthropometric somatotype the samples differed in all three somatotype components (endomorphy, mosomorphy, ectomorphy), the greatest difference being in ectomorphy. The results obtained are recommended for use in the primary preventive care of coxarthrosis.

Adult↗

Amyloidosis in rheumatoid arthritis. A study of 48 histologically confirmed cases.

In a group of 269 patients with rheumatoid arthritis histological examination demonstrated amyloidosis in 48 cases, viz. in 7 post mortem cases, in 28 rectal biopsies, in 12 renal biopsies and in one liver biopsy. Examination for amyloidosis was carried out in all patients who had proteinuria, otherwise in non-selected patients with definite rheumatoid arthritis. Even through rectal biopsy is a valuable screening method, it should not be overestimated, because in 12 patients with renal biopsy positive for amyloid, the foregoing rectal biopsies had been negative. According to our experience the most valuable method for diagnosis of amyloidosis in rheumatoid patients is renal biopsy, whereas synovial biopsy is the least conclusive. This paper, according to our knowledge, is the first report on observations of a regression of the inflammatory activity of rheumatoid arthritis and nephrotic syndrome as well as a complete morphological regression of amyloidosis.

Adult↗