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Biomedical subjects

M I Bakanov

Publications and source records attributed to M I Bakanov.

At least 19 recordsLinked to original sources

[Lipid peroxidation in neurological pathology in children].

The intensity of lipid peroxidation (LPO) was studied by the content of the listed below in erythrocytes of children with neurological pathology: diene conjugates, trienketones and LPO products. Intensified LPO processes were registered in children with neurology and primarily in those with cerebral spastic infantile paralysis (CSIP). The detected changes in the LPO products of erythrocyte membranes depended on a disease variation on an intellectual development of sick child. The LPO activation affects possibly the development (lesion) of craniocerebral innervation in patients. Changed LPO parameters were found in examined sick children to be most closely related with the development of their motor skills and with the formation of the bone-muscle system, thus, an essentially higher content of LPO products was registered in erythrocytes of children with a reduced muscular activity (in those not capable of sitting, standing or walking). Considering the above stated and with respect to the deficit of Vitamin E registered in such patients, children with CSIP or with impending CSIP need, obviously a prolonged membrane-stabilizing therapy by antioxidants and essential phospholipids including traps of radicals, which should cut the number of children with severe CSIP and with residual perinatal lesions of the central nervous system.

Biomarkers↗

[Biochemical changes of red blood cells in children with cerebral palsy and other organic brain disorders].

One hundred and twenty-five children with cerebral palsy (n = 108) and organic CNS lesions (n = 17) have been studied. A control group included 30 children without any neurological lesions. Lipids and phospholipids spectra, Ca, Mg- and Na, K-ATPases, intensity of red blood cells lipid peroxidation and erythrocyte free magnesium content have been determined. Children with cerebral palsy had a higher percentage of cholesterol ethers and final products of lipid peroxidation in red blood cells--Shiff bases and a decrease of intraerythrocytic free magnesium. Patients with atonic-astatic syndrome were characterized by a sharp increase of Na, K-ATPase activity accompanied by increased content of such phospholipids as cardiolipin and phosphatide acid as well as Shiff bases. In patients with organic CNS lesions, Shiff bases content was similar to that in the control group, intracellular magnesium also did not differ significantly from the controls, but cardiolipin, phosphatide acid, cholesterol ethers and triglycerol concentrations were reduced. All the patients with neurological disorders had an increased percentage of phosphatidilcholin in red blood cells.

Adenosine Triphosphatases↗

[Red cell phospholipid composition in children with neurological pathology].

Changes of red cell membrane phospholipid composition were studied in children with different neurological disorders--cerebral palsy, organic CNS damages and perinatal encephalopathy. The percentages of phosphatidylcholin and, in some cases, of phosphoglycerides increase depending on the disease type. The above changes were determined by craniocerebral innervation disturbances and concerned mainly the content of phosphatidylcholin, phosphatidylserine and phosphatidylethanolamin. The determination analysis established that the changes of erythrocytes phospholipid content correlated with clinical state severity and intellectual development of children, and to depend on the disease type and may affect directly development/disturbance of craniocerebral innervation and intellectual development. The age changes were shown to influence phosphatid acid percentage in the patients.

Brain↗

[Disorders of membrane mechanisms in stressor damage to the heart in rats of various strains].

The resistance of the rat's organism to emotional stress is determined by alterations of the heart's electric stability. The condition of lipid oxidation is accompanied by alterations of cholesterol and ionised calcium concentrations. Such alterations were expressed in Fisher rats to a greater extent. It conformed to the morphophysiological peculiarities of this group of animals and confirmed its considerable species sensibility.

Animals↗

[State of lipid peroxidation processes and cyclase system components in lymphocytes and blood plasma of children with aplastic anemia].

Lipid peroxidation, antioxidant defence enzymes in lymphocytes and the content of cyclonucleotides in blood plasma were examined in children (3 groups) with aplastic anemia. The groups were formed depending on the disease stages in the examined. Disorders in the distribution of the activity of antioxidant defence enzymes, in the content of hydroperoxides and malonic aldehyde were discovered, with these disorders being diverse and dependent on the disease stage. The cAMP/cGMP in blood plasma was changed towards considerable rise of the cAMP level. During a remission, a majority of the characteristics did not return to normal.

Adolescent↗

[Interrelation of changes in the levels of calcium, calmodulin and cyclic nucleotides in children with ectopic types of arrhythmia].

In 61 children with different patterns of ectopic arrhythmias (premature heart beat, paroxysmal and non-paroxysmal tachycardia), the peripheral blood leukocytes and myocardial tissues were examined for the content of calmodulin (CM), ionized calcium (Ca2+) and cyclic nucleotides (CN). The data obtained indicate that the CM-Ca2+ and CN system are involved in the genesis of arrhythmias in children and may be used as additional criteria in the diagnosis of cardiac rhythm abnormalities.

Adolescent↗

[Clinical significance of cyclic nucleotide metabolism and calcium level in children with arrhythmia].

The cyclic nucleotides (CN) system and Ca2+ content were examined in children with various heart rhythm disorders (HRD). Marked changes were demonstrated which correlated to clinical manifestations of the arrhythmias: their type, phase, duration and favorable changes in their course. Antiarrhythmic therapy was associated with a normalization of the altered levels of cyclase components and Ca2+. The results suggest that intracellular metabolism regulation may be disrupted in HRD cases and play an important role in the development of arrhythmias.

Adolescent↗