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Biomedical subjects

M Iskandarani

Publications and source records attributed to M Iskandarani.

6 recordsLinked to original sources
PubMed1985

Mirizzi syndrome.

A 56-year-old woman was admitted to the hospital because of icterus and pruritus of 1 month duration. During her evaluation, endoscopic retrograde cholangiography revealed large cystic duct stones causing partial obstruction of the common hepatic duct. Successful management was accomplished with choledochojejunostomy and T-tube decompression. The case herein reported illustrates the typical clinical presentation and radiographic features of the Mirizzi syndrome.

Cholangiopancreatography, Endoscopic Retrograde↗
PubMed1984

Successful medical management of a Yersinia enterocolitica liver abscess.

An insulin-dependent diabetic patient with liver abscess secondary to Yersinia enterocolitica without septicemia is described. He had the diagnosis established by transhepatic needle aspiration of the abscess cavity. Successful management was accomplished with antibiotic therapy alone.

Anti-Bacterial Agents↗
PubMed1984

Bilateral nipple necrosis after intravenous vasopressin therapy.

Bilateral nipple necrosis developed in two patients after intravenous vasopressin therapy for bleeding esophageal varices. When vasopressin therapy was promptly tapered and discontinued, recovery of skin changes occurred gradually. The role and some of the reported complications of vasopressin therapy in bleeding esophageal varices are discussed herein.

Adult↗
PubMed1984

Focal nodular hyperplasia associated with the use of a synthetic anabolic androgen.

An adolescent with stunted growth was treated for 6 months with a synthetic anabolic androgen. During his evaluation for right upper quadrant pain an abnormal liver scan demonstrated what proved to be a hypervascular hepatic lesion. At surgery, the diagnosis of focal nodular hyperplasia was established. Herein we document the unusual association of focal nodular hyperplasia with use of a synthetic anabolic androgen.

Child↗
PubMed1981

Kinetics of calcium transport in the hamster duodenum and ileum.

Dietary calcium is absorbed chiefly from the small intestine, and characteristics of the transport process differ depending on site. Because transport behavior is crucial to calcium homeostasis, we characterized kinetics of duodenal and ileal calcium transport in vivo in the hamster. We measured net calcium transport with 40Ca and flux into the animal (influx) with 45Ca over the concentration range of 0- 25 mM. Nonmediated transport was twice as great in duodenum as in ileum. Mediated transport showed Michaelis-Menten kinetics. Kt for mediated transport (4.3 mM) was the same in duodenum and ileum, whereas Vmax (mumol/h per g) was twice as great in ileum (290) as in duodenum (137). Thus, Kt defines similar carrier function at both sites, but Vmax suggests calcium transport capacity is doubled in ileum. The lower nonmediated transport in ileum limits secretion and the higher Vmax absorbs calcium at a greater rate, permitting the ileum to move luminal calcium against a concentration gradient more effectively than the duodenum. Thus, ileal calcium transport, by its site and functional characteristics, makes a crucial contribution to calcium homeostasis in the hamster.

Animals↗