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Biomedical subjects

M K Hariprasad

Publications and source records attributed to M K Hariprasad.

14 recordsLinked to original sources
PubMed1982

Dialytic ascitic ultrafiltration in refractory ascites.

Nine patients with hepatic cirrhosis and refractory ascites underwent dialytic ascitic ultrafiltration with intraperitoneal reinfusion of the concentrated ascites. Ascites was removed via a peritoneal catheter and ultrafiltered using a hemodialyzer. The concentrate was continuously returned to the peritoneal space. A mean of 4.8 l of protein-free ascitic ultrafiltrate was removed without adverse effects over the course of the 3- to 5-h procedure. Preliminary evidence indicates a potential role for dialytic ascitic ultrafiltration in the management of refractory ascites.

Ascites↗
PubMed1981

Hemodialysis for uremic schizophrenics: no psychiatric improvement.

Two uremic schizophrenics received 73 and 26 hemodialyses in 33 and 18 weeks respectively. Serial objective psychiatric scales were applied and indicated lack of improvement of schizophrenia during dialysis therapy. Further trials with hemodialysis on uremic schizophrenics need to be undertaken before this investigational therapy is offered for non-uremic schizophrenics.

Humans↗
PubMed1981

Urinary osmolality in lithium and non-lithium treated psychiatric patients.

A study was conducted to determine the prevalence of urine concentrating defect in 57 psychiatric patients who were treated with lithium and 42 patients who were not treated with lithium. Analysis of results indicated that there was no statistically significant difference between the lithium treated and control groups in their random 12 hour fluid deprivation urine osmolality, 0.5 greater than p greater than 0.1, t-test. The authors suggest that impaired urinary concentrating ability occurs frequently in both lithium and nonlithium treated psychiatric patients. This isolated finding alone is insufficient to warrant the diagnosis of lithium nephrotoxicity.

Diabetes Insipidus↗
PubMed1980

Hyponatremia in psychogenic polydipsia.

Twenty psychotic patients with psychogenic polydipsia had hyponatremia (98 to 124 mEq/L) lasting up to 28 months, with headache, hypertension, dementia, seizures, lethargy, and coma. Two deaths also may be attributed to this syndrome. Patients drank 7 to 43 L of water daily. Urine was dilute during this water load (37 to 95 mOsm/kg), and free water clearance ranged from 12 to 36 L/day, while plasma osmolality was 236 to 244 mOsm/kg. During fluid deprivation in seven such patients, urinary osmolality exceeded plasma osmolality when plasma concentration had risen to between 242 and 272 mOsm/kg, thus suggesting a "reset osmostat" or antidiuretic hormone response to nonosmotic stimuli. This tended to sustain hyponatremia. Polydipsia should be recognized as a cause of hyponatremia, perhaps with reset osmostat. This ultimately may cause dementia or death, possibly secondary to recurrent cerebral edema. This sequence of events is potentially preventable or correctable.

Adult↗