Recognizing dental malocclusion in children.
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Biomedical subjects
Publications and source records attributed to M L Helpin.
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Michelin Tire syndrome is a genetic disease caused by smooth muscle hematoma formation, a condition which causes symmetrical, ringed creases around all extremities and the torso. Oral findings in a child with this syndrome were unusual and are presented in this case report.
Several resin-modified, glass-ionomer cements have proved successful when used in Class III and Class V cavities in primary teeth. One of them, Vitremer, is proving to be very durable when used in Class I and Class II cavities in primary teeth. The material is handled as quickly and easily as silver amalgam. The components of the Vitremer Tri-Cure System are: Primer, Glass powder, Liquid, and Resin gloss. In addition to a light-curing mechanism, Vitremer hardens by the acid base, glass-ionomer reaction, and a chemical "dark cure", catalyzed by a reduction/oxidation reaction. Over 600 Class II Vitremer restorations have been placed and over 250 have been in the teeth long enough (12 to 18 months) to be observed regularly.
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The light-hardened glass ionomer/resin dental restorative material was introduced in 1992. Its formula includes 80 percent glass ionomer material combined with a 20 percent visible light polymerized resin component. The authors have completed over 1300 class I, class III, and class V restorations in primary teeth, using the new material. The new material is considered by the authors to be a great improvement over the self-hardening glass ionomers. The addition of the 20 percent light-hardened resin component to the glass ionomer formula gives the material an initial setting time under sixty seconds, and improves resistance to wear and fracture.
Fourteen hydrocephalic children with ventriculo-peritoneal shunts received routine dental prophylaxis and topical fluoride application. No antibiotics were administered to these children for any reason during the three months before treatment or during the twelve months after treatment. None of these children presented with any signs of shunt infection during the twelve-month posttreatment period. In spite of the small sample size, this prospective pilot study suggests that patients with ventriculo-peritoneal shunts are not susceptible to shunt infection following a bacteremia induced by a dental prophylaxis and topical fluoride treatment. Dental prophylaxis without antibiotic coverage in patients with V-P shunts, therefore, does appear safe. We recommend that further study with a larger population, or a collaborative study by several medical centers, be performed to establish more conclusively that prophylactic antibiotics are not necessary for patients with ventriculo-peritoneal shunts who receive dental procedures. In addition, other investigations are needed to determine the risk of shunt infection with more invasive dental procedures, such as periodontal surgery or tooth extraction.