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M Rossleigh

Publications and source records attributed to M Rossleigh.

5 recordsLinked to original sources
PubMed2001

TI-201 scintigraphy in muscle trauma.

Muscle uptake of Tl-201 thallous chloride in the larger or more major groups, particularly the legs, chest, and upper limb areas, is not uncommonly observed in healthy persons. However, as illustrated in three patients, such uptake, when localized or asymmetric, may provide evidence of unrecognized traumatized muscle, resulting from more chronic or subacute muscle overuse than the acute muscle insult that produces the marked uptake of Tc-99m phosphates associated with frank rhabdomyolysis.

Adult↗
PubMed1995

Intrahepatic bile duct dilatation and cholestasis in autosomal recessive polycystic kidney disease. Demonstration with hepatobiliary scintigraphy.

Fifteen patients with the clinical and radiologic features of autosomal recessive polycystic kidney disease were investigated with radionuclide hepatobiliary scintigraphy. In nine patients (60%), cholestasis and intrahepatic bile duct dilatation were demonstrated. A 10th child had scintigraphic evidence of cholestasis, but the bile ducts were not dilated. The other five children has normal h hepatobiliary scans. The authors conclude that intrahepatic bile duct dilatation with cholestasis (Caroli's disease) is part of the clinical spectrum of autosomal recessive polycystic kidney disease and that hepatobiliary scintigraphy can be of value in determining the extent of hepatobiliary disease in this group of patients.

Bile Ducts↗
PubMed1991

Abnormal cortical appearances in pediatric renal allografts.

Renal cortical studies were performed in 19 children with renal transplants. There were 10 normal studies and 9 abnormal studies, 8 of which showed multiple large focal peripheral cortical defects. The following factors showed a positive correlation: (a) the ischemia time of the transplant kidney was significantly shorter in patients with normal studies; (b) cadaver grafts were more likely to have abnormal scan appearances than living related donor grafts; and (c) in four of the five patients with double renal arteries, the scans were abnormal in multiple sites. A possible pathophysiologic mechanism to explain these scan appearances is asymptomatic segmental graft infarction secondary to progressive vascular disease. These infarcts may be a long-term sequela of ischemic insult at the time of or prior to the insertion of the renal allograft.

Child↗