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M Straschill

Publications and source records attributed to M Straschill.

At least 37 records · Page 2Linked to original sources
PubMed1977

Discharges of superior colliculus neurons during head and eye movements of the alert cat.

452 single neurons from the superior colliculus were recorded in awake and non-paralysed cats. 75 neurons were obtained from cats with unrestrained horizontal head movements. 228 neurons remained unaffected by saccadic eye movements. Eye movement related discharge followed the onset of saccades in 156 neurons either only in the presence of a visual pattern (92 neurons) or in darkness, too (64 neurons). The latter reaction type probably depends on eye muscle afferents. In 48 neurons eye movements related activity preceded the onset of eye movements. 12 neurons eye movement related activity preceded the onset of eye movements. 12 neurons fired in synchrony with eye movements of any direction (type I). 30 neurons were excited during contralaterally directed eye versions within or into the contralateral head related hemifield. They were inhibited when the eyes moved within or into the ipsilateral head related hemifield (type II). 6 neurons with constant maintained activity during fixation were inhibited by ipsilaterally directed saccades, but remained unaffected by contralateral eye movements. Head movement related discharge followed the onset of head movements in 20 neurons only in presence of a visual pattern and also in darkness in 6 neurons. Ipsilateral head movements or postures strongly suppressed maintained activity and visual responsiveness of some neurons. 15 neurons discharged in synchrony with and prior to contralateral head movements. Ipsilateral head movements inhibited these neurons. Activation or inhibition were usually related to movement and to posture, exceptionally to movement or to posture. Electrical stimulation of recording sites of these neurons through the recording microelectrode elicits contralateral head movements.

Animals↗
PubMed1977

[Electromyographical and clinical follow up study of brachial plexus lesions (author's transl)].

21 cases of brachial plexus lesions were reexamined clinically and electromyographically after a posttraumatic interval of 3 to 11 years. In accordance with Brooks (1) recovery--as judged by muscle strength, sensation and amount of EMG activity during volontary action--was relatively good in upper plexus lesions, fair in middle plexus lesions and very poor in lower plexus lesions (Fig. 1 A, B). Even in cases of incomplete lower plexus lesions with small residual innervation initially, recovery was only moderate (Fig. 1 C). The tendency for reinnervation decreased with increasing distance of target muscles from the lesion site in the plexus (Fig. 2). Motor and sensory deficits in corresponding dermaresp. myotomes were either congruent or more frequently incongruent with prevalence of motor deficits (Fig. 3). Earliest electromyographical signs of reinnervation were observed after 4 to 9 months (upper and middle plexus lesion). Reinnervation of proximal muscles was completed after 11 months to 2 years (Fig. 4). Posttraumatically regenerated nerve fibers had often decreased conduction velocities (some values as low as 7 m/sec) and showed sometimes abnormal target muscles leading to paradoxical innervation and synkinesias between antagonistic muscles.

Brachial Plexus↗
PubMed1975

The effects of a spirolactone derivative on EEG and cortical single unit activity in the cat.

With systemic (60-150 mg/kg) and topical application, Aldactone exhibited strong convulsant action on the cat cerebral cortex. The most common abnormal interictal features of the surface ECoG pattern were sequences of fast waves and slow negative waves associated with positive sharp waves, negative field potentials and burst-like neuronal activation in the extracellular microelectrode record. Another abnormal pattern was a period of ECoG and neuronal inactivity subsequent to the negative transients. These series of sharp and slow waves and of inactivity were interrupted by tonic and clonic ECoG seizures characterized by regular surface positive spikes. Development of seizures, but not of interictal phenomena, could be prevented by intravenous application of Diazepam (1 mg), Nembutal (30 mg/kg) and--less effectively--by Phenhydane (20 mg/kg).

Administration, Topical↗