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Mehmet Ateş

Publications and source records attributed to Mehmet Ateş.

5 recordsLinked to original sources
PubMed2005

A novel reporting approach to coronary angiography: "segmental coding system".

BACKGROUND: A new systematic reporting system for coronary angiography has been developed, which is capable of describing any visible intraluminal or extraluminal conditions with the exact coordinates. METHODS: In this method, called "segmental coding system"(SCS), the part of the artery that is located between its two subsequent branches is considered to be an "angiographic segment". Conditions are localized according to their relationship with these angiographic segments and the anatomic border of the segments (coronary ostiums, primary, secondary and tertiary branches, grafts and proximal and distal anastomosis sites). They are also described by using a special coding system that consists of letters, numbers and signs. SCS can supply the name (stenosis, occlusion, contour deformity, aneurysm, rupture, anatomical variation, existence of stent, etc.) and the exact localization (coordinates) of the condition with its properties; filling direction, and the collateral system that fills the vessel. RESULTS: We applied SCS to more than 500 cineangiograpies. According to our experience, SCS provides more objective, detailed, and even correct information than the current narrative reporting system. SCS also offers many extra advantages. (a) It can describe all imaginable types of lesion combinations. (b) All of the existing conditions can be listed without missing. (c) The definitions are very precise and clear. They can easily be understood by everyone in the same way. (d) It is more advantageous on archiving, searching the database, and comparing the subsequent reports for the same patient. (e) In the future, by using specially tailored software, personal and detailed angiographic images will be reproduced from the SCS data. CONCLUSIONS: By being introduced into clinical practice, we believe, SCS will prove a very useful tool for both surgeons and cardiologists.

Coronary Angiography↗
PubMed2003

Two findings of portal hypertension: evaluation of correlation between serum-ascites albumin gradient and esophageal varices in non-alcoholic cirrhosis.

BACKGROUND/AIMS: Esophageal varices and serum-ascites albumin gradient (SAAG) are two major findings of portal hypertension. Recently, correlation between these two findings in patients with cirrhosis due to alcohol has been attracted attention. We aimed at evaluating whether a correlation exists between these parameters in the patients with non-alcoholic cirrhosis. METHODS: Albumin levels in the serum and ascites and esophageal varices were studied and the correlation between these parameters was assessed in 45 patients with non-alcoholic cirrhosis detected between January 2002 and June 2003. RESULTS: Thirty-two of the patients were male and 13 female. The average age of the patients was 56.3+/-12.5 years (range 22-85 years). The causative agents were found to be hepatitis B virus in 35 patients and hepatitis C virus in six patients; no etiology could be determined in the remaining four patients. Serum level of albumin was determined as 2.53+/-0.53 g/dl, ascites level of albumin as 0.42+/-0.31 g/dl and SAAG as 2.1+/-0.51. Endoscopic esophageal examination revealed first-degree esophageal varices in 15 patients, second-degree esophageal varices in 18 patients and third-degree esophageal varices in eight patients; no esophageal varices could be found in four patients. There was no correlation between the degree of the esophageal varices and serum levels of albumin (p=0.7) and SAAG (p=0.2); but a weak correlation was found between the degree of the esophageal varices and ascites levels of albumin (p=0.03, r=0.30). Furthermore, the patients were classified by their SAAG values, and their varices were then assessed. Two of four patients with SAAG values between 1.1 and 1.49 had esophageal varices, as did 13 of 15 patients with SAAG values between 1.5 and 1.99, and all of the patients with SAAG values greater than 2.0. CONCLUSION: All SAAG values were greater than 1.1 in our non-alcoholic cirrhosis cases. The correlation that has been found to exist between SAAG and esophageal varices could not be found in our patients with non-alcoholic cirrhosis. It is remarkable that most of the patients with non-alcoholic cirrhosis presenting with ascites and all of the patients with an SAAG value greater than 2.0 had esophageal varices.

Adult↗