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Biomedical subjects

N B Perepech

Publications and source records attributed to N B Perepech.

30 records · Page 2Linked to original sources
PubMed1989

[Oxygen supply of the body in myocardial infarction].

Oxygen transport and demand were examined in 71 patients with myocardial infarction within the first 5 weeks of the disease. The volume of blood-transported oxygen was shown to be determined mainly by the blood circulatory minute volume. Lower arterial blood oxygen is an additional factor of diminished oxygen transport in hypodynamic central hemodynamics. Within the first 5-10 days of myocardial infarction, there was a progressive reduction in oxygen transport, but by days of 28-35, there was its gradual increase. These changes are the most prominent in patients with a hyperdynamic type and negligible in those with a hypodynamic type within the first 24 hours of myocardial infarction. The important of oxygen transport decrease compensation is its increased tissue extraction from arterial blood, thereby providing the stable oxygen demand in the wide range of its supply fluctuations.

Adult↗
PubMed1986

[Changes in stroke volume in ventricular asystole].

Change in the stroke volume in ventricular extrasystoles was studied in 27 patients. In patients with cardiac insufficiency a decrease in the extrasystolic stroke volume was more noticeable than in patients without cardiac insufficiency. "Hemodynamic debts" for extrasystolic and postextrasystolic contractions in patients with the presence and absence of cardiac insufficiency were undistinguished. In interpolated extrasystoles "hemodynamic debts" were less than in non-interpolated.

Adult↗
PubMed1986

[Features of the hemodynamics of myocardial infarct patients undergoing the most widely used rehabilitation programs].

Integrated body rheography, radiocardiography and radionuclide ventriculography were used to investigate hemodynamic changes in 101 myocardial infarction patients during the hospital stage of the disease. Changes in major hemodynamic parameters were demonstrated by the end of the 1st week and when walking was resumed. At the resumed-walking stage, the mechanism of declining stroke and cardiac indices was shown to depend on physical activation rates. Hemodynamic response is mostly conditioned by myocardial insufficiency when walking is resumed rapidly during the 2nd week, and by smaller venous return due to hypovolemia where it is resumed slowly during the 4th week. Expanding motion regimens at slow rates results in persistent hemodynamic disturbances in myocardial infarction patients.

Adult↗