Cardiopulmonary responses to selective increase in cephalic and spinal CSF pressure.
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Animals↗
Biomedical subjects
N C Gonzalez
Publications and source records attributed to N C Gonzalez.
Cardiopulmonary responses to uniformly elevated CSF pressure.
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Animals↗
Primary negative inotropic effect of tris(hydroxymethyl)aminomethane.
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Acid-Base Equilibrium↗
Circulatory effects of moderately and severely increased intracranial pressure in the dog.
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Blood Pressure↗
Cardiovascular response to independent cephalic and spinal cord pressure elevators.
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Animals↗
Mechanism of action of tris (hydroxymethyl) aminomethane on the negative inotropic effect of carbon dioxide.
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Animals↗
Contractility in isolated mammalian heart muscle after acid-base changes.
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Animals↗
[Effect of the reduction of extracellular sodium concentration on contractility].
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Animals↗
Extracellular vs. intracellular pH as a determinant of myocardial contractility.
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Acid-Base Equilibrium↗
Lack of effect of Tris-(hydroxymethyl)aminomethane upon myocardial contractility.
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Animals↗
Myocardial potassium uptake with constant arterial potassium concentration.
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Animals↗
Blood viscosity in chronically hypoxic rats: an effect independent of packed cell volume.
We compared apparent blood viscosity, measured with a cone-plate viscometer, in normoxic and chronically hypoxic rats. All comparisons were made at equal packed cell volume (PCV) and shear rate conditions. Apparent viscosity of whole blood from the hypoxic rats was significantly lower than that from normoxic littermates and was similar to that of young rats. Apparent viscosity of red cells from hypoxic rats suspended in phosphate buffered saline remained lower than that from the normoxic rats. When blood cells from the hypoxic rats were suspended in plasma from normoxic rats, apparent viscosity was lower than when blood cells from normoxic animals were suspended in plasma from hypoxic rats. The lower viscosity of the blood from hypoxic rats appears to be associated with characteristics present in newly generated red cells. The reduced apparent viscosity of blood in hypoxic rats may partially compensate for the increase in PCV, at least during the early stages of hypoxia.
Aging↗