Biomedical subjects

N Greenidge

Publications and source records attributed to N Greenidge.

5 recordsLinked to original sources

PubMed1991

Vascular anatomy of the fifth metatarsal.

The extraosseous and intraosseous vascular anatomy to the fifth metatarsal as visualized in a group of below-the-knee amputation specimens has been described. The extrinsic circulation to the area is provided by the dorsal metatarsal artery, the plantar metatarsal arteries, and the fibular plantar marginal artery. These three source arteries supply branches to the metatarsal and adjacent joints. The intraosseous vascularity consists of a periosteal plexus, a nutrient artery, and a system of metaphyseal and capital vessels.

Arteries↗

PubMed1990

Vascularity of the posterior tibial tendon.

Operative exploration of ruptures of the posterior tibial tendon has revealed a relatively constant site of damage just posterior and distal to the medial malleolus. It has been hypothesized that the stress that is applied to the tendon as it passes under the medial malleolus may compromise the vascularity of the tendon in this area, a process that is similar to that described for the supraspinatus tendon. In order to evaluate this possibility, we studied the vascularity of the posterior tibial tendon. We injected twenty-eight cadaveric limbs with an India ink-gelatin mixture and cleared them using a modified Spalteholz technique. The gross external and internal vascularity were drawn, and the specimens were photographed. The vascularity of the tendons was noted to be abundant at the osseous insertion and the musculotendinous function of the tendon. There was a zone of hypovascularity posterior and distal to the medial malleolus in all specimens. The results of this study suggest that relative avascularity and resulting degenerative changes may be predisposing factors in rupture of the posterior tibial tendon, and that they have important implications in terms of the operative management of the rupture.

Adult↗

PubMed1987

Normal trunk muscle strength and endurance in women and the effect of exercises and electrical stimulation. Part 1: Normal endurance and trunk muscle strength in 101 women.

The lack of trunk muscle strength and endurance has frequently been cited as a suspected factor in the etiology of low-back pain. Several investigators have suggested that asymptomatic patients have stronger trunk muscles than patients with low-back pain. People who are physically fit appear to have a decreased incidence of low-back pain. Increased trunk muscle endurance also have been observed to decrease the incidence of low-back pain. The objective evaluation of the strength and endurance of trunk musculature may, therefore, be significant. Part 1 of this study was designed to develop a reproducible strength-endurance screening procedure and to establish normal isometric-isokinetic trunk muscle strength and endurance parameters for women. This study showed that isometric trunk flexion varied from 19-109 Nm and trunk extension from 38-168 Nm. Peak values for isokinetic trunk flexion at two speeds (30 degrees per second and 60 degrees per second) varied from 17-191 Nm and isokinetic trunk extension from 14-208 Nm. The average endurance time for trunk extensors tested with the Sorensen test was 196 seconds.

Adolescent↗

PubMed1986

Spinal configuration during lifting.

The change in spinal configuration of the cervical, thoracic, and lumbar regions in relation to an amount of weight lifted was determined using videophotogrammetry. Fifteen healthy male subjects, 20-38 years of age, with no previous history of back pain participated in the study. The subjects lifted a crate containing 0, 10, and 20 kg weights using the straight-legs, bent-over-back method of lifting. The results showed that cervical and thoracic spinal segment configurations were not significantly influenced by the amount of weight lifted and that the mobility of the lumbar spinal segment was significantly decreased with increasing load (p = .03).

Adult↗

PubMed1976

Inhibition of actions of glucagon in adipocytes by gastric inhibitory polypeptide.

Possible interactions between gastric inhibitory polypeptide (GIP) and glucagon were investigated in rat adipocytes. GIP was nonlipolytic and inhibited lipolysis stimulated by glucagon but not that stimulated by secretin or vasoactive intestinal polypeptide (VIP). GIP competed with 125I-glucagon for binding to adipocyte receptors, and at physiologic concentrations inhibited the stimulation of AMP produced by glucagon. Thus GIP acts as an inhibitor of actions of glucagon on adipocytes and may be a physiologic modulator of effects of glucagon.

Adipose Tissue↗