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Biomedical subjects

N I Vasilenko

Publications and source records attributed to N I Vasilenko.

At least 19 recordsLinked to original sources
PubMed1992

[Local treatment of suppurative wounds].

The authors treated 327 patients with purulent wounds, using a solution containing dimexide, iodine, tannin, and as well the flowing-aspirating drainage and silver nitrate electrophoresis. Rapid cleaning of the infected wounds and shortening of duration of the in-patient treatment were noted.

Adult↗
PubMed1977

[Diagnosis and treatment of complicated pancreatic cysts].

The results of the treatment of 18 cases with complicated pancreatic cysts (11 men and 7 women) were studied. Good results were obtained after cystectomy and resection of the pancreas together with the cyst, when the latter was uncomplicated. With complications, external and more seldom internal drainage were preferred. In the postoperative period 3 patients developed persistent pancreatic fistula, one patient developed malignization of the cyst and another one -- thrombosis of the splenic vein.

Adolescent↗
PubMed1976

[Complications of benign nonepithelial tumors of the gastrointestinal tract].

The results of observations over 8 patients with complicated forms of benign nonepithelial tumors of abdominal organs are reported, including leiomyoma--in 7 cases and neurinoma--in 1. Isocomplications of this group of tumors in 5 patients were as follows: invagination of the small intestin--in 1, perforation and malignant transformation of the tumor--in 1. The patients were treated surgically. Resection of the organ involved seems to be preferable.

Adult↗
PubMedDate not supplied

[The importance of the state of the oxygen balance in the early period following severe mechanical trauma in the pathogenesis of the shock lung syndrome].

To elucidate the role of oxygen balance disturbances in the pathogenesis of shock lung syndrome (SLS) central hemodynamic parameters, arterial and mixed venous blood content, O2 transport (TO2) and consumption (VO2), TO2 reserve have been studied in 95 patients after trauma. Two groups of patients have been identified: group I--control (n = 48); and group II--patients with SLS (n = 47). Significant differences in central hemodynamics and oxygen balance between the groups before the manifestation of SLS symptoms were lower values of the cardiac output, TO2, VO2 and TO2 reserve in patients of group II. If, on the first day after trauma, there was hyperdynamic type of circulation and 40-50% increased (as compared to the physiological norm) VO2 in combination with sufficient TO2 reserve (about 100 ml/(min.m2) the likelihood of SLS onset was considerably reduced.

Adolescent↗
PubMedDate not supplied

[Characteristics of the functioning of the oxygen supply system on the 1st day following trauma in patients with various courses of the post-traumatic period].

A correlation between a number of oxygen balance parameters and the application of regression analysis methods has been studied in 67 patients within the first 24 h after trauma. The regression equations demonstrate that O2 transport (TO2) reserve sufficient to meet high metabolic requirements in the first 24 h after trauma promoted prompt normalization of the vital body functions and a more favourable course of the posttraumatic period. The formula has been derived and a nomogram has been plotted for the calculation of TO2 reserve in the first 24 h after trauma. It has been found that, with TO2 reserve less than 50 ml/(min.m2), the likelihood of complications in the posttraumatic period and the lethal outcome, especially in patients with acute respiratory failure is increased. TO2 less than 360 ml/(min.m2) is unable to ensure high O2 consumption and is prognostically unfavourable in patients with severe mechanical trauma.

Adolescent↗
PubMedDate not supplied

[Aspects of pathogenesis of "shock lung" in patients with massive blood loss and trauma in the early postresuscitation period].

116 patients with massive blood loss and trauma have been studied in intensive care units. It has been established that the main reason for the onset of hypoxemia in "shock lung" is disturbed gas exchange in the lungs due to an expanded physiological dead space (VD/VT) and blood shunting in the lungs (QS/QT). Intravascular blood coagulation is one of the main reasons for the development and maintenance of systemic hypoperfusion in the lung tissue. Progressive increase in venous admixture during inhalation of 100% oxygen over a five-day treatment period is due to an increased number of still blood-supplied but already unventilated affected alveoli.

Hemorrhage↗