PubMed Health⌕ Search

Biomedical subjects

O Pereni

Publications and source records attributed to O Pereni.

5 recordsLinked to original sources
PubMedDate not supplied

[The technical modalities of duodenojejunoplasty in large parietal defects of the duodenum].

The authors present two technical modalities for solving extensive defects of the duodenal wall which occurred during surgery for large duodenal ulcers, which in one case involved the entire anterior part of D1-D2. In another case the duodenal wall defect was due to tumoral invasion by cancer of the hepatic angle of the colon. The solution consisted in duodenoplasty with excluded intestinal loop in "Y", in the first case, and duodenoplasty on an omega loop associated with GEP in the second case.

Adult↗
PubMedDate not supplied

[Post-traumatic acute pancreatitis].

In connection with 7 observations of acute traumatic pancreatitis the authors review the data published in the specialized literature as well as their personal experience. A series of aspects are discussed, such as the frequency, the mechanisms involved in the determination of the phenomena, clinical and diagnostic aspects, as well as the therapeutic attitude. The importance of functions is stressed, combined with peritoneal flooding, and dosage of amylases in the fluid obtained in view of making an early diagnosis, and for an efficient therapy, in most of the cases of the surgical type.

Abdominal Injuries↗
PubMedDate not supplied

[Pathogenetic aspects of acute renal insufficiency in experimental acute pancreatitis].

The authors carried out a study of some pathogenic mechanisms related to the development of acute renal failure in animals following pancreatitis-induced shock. The study was made in 110 white rats in which acute pancreatitis was induced experimentally, and the results were compared with those obtained in a control lot of 40 rats. The behaviour of some biochemical parameters was investigated (cathecholamines, acid phosphatases, catepsine, aminoacids and polypeptides), in tissue homogenates (liver, intestine and kidney), and in the serum. Histopathologic changes were also evaluated, which occurred in the intestine, the kidney, the liver and the pancreas. Histopathologic changes in the kidney, and acute renal failure which accompanied them in rats with acute pancreatitis are rather a result of protesic activation in the tissues of the organs involved (especially the intestine and the kidney), and are less the results of enzymatic extension or metastases following pancreatic necrosis.

Acute Disease↗