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Biomedical subjects

P J Koehler

Publications and source records attributed to P J Koehler.

17 recordsLinked to original sources
PubMed1992

Late posttraumatic nonvascular pulsating eye.

A patient is reported with unilateral intermittent pulsating eye caused by the combination of an orbital encephalocele and hydrocephalus, thirty four years after a head injury. Treatment of the hydrocephalus resulted in disappearance of the eye pulsations and encephalocele, as well as in improvement of the gait.

Cerebrospinal Fluid Shunts↗
PubMed1991

Chiari's description of cerebellar ectopy (1891). With a summary of Cleland's and Arnold's contributions and some early observations on neural-tube defects.

One hundred years ago, Chiari published his first paper on cerebellar ectopy. The contribution of his paper and those of Cleland (1883) and Arnold (1894) to the development of the knowledge of craniocervical abnormalities is discussed. Some early contributions (those of Tulp and Ollivier d'Angers) to the subject of neural-tube defects were also studied and it is concluded that Chiari's name should be connected with the different types of cerebellar ectopy.

Arnold-Chiari Malformation↗
PubMed1988

[Bacterial meningitis in the aged. Pitfalls in medicine].

Bacterial meningitis in aged patients is a diagnosis that can be difficult to make. Three case-histories, one of them being diagnosed non-purulent bacterial meningitis, are presented to demonstrate this. If classic symptoms and signs are being absent, evidence of an infection should suggest meningitis, if other common infections, like urinary tract or respiratory infections, are excluded, and even in these cases a concomitant meningitis may be possible. Even then a high mortality should be taken into account. Only tenacity at diagnostics will be able to reduce this.

Aged↗
PubMed1986

The Brown-Séquard syndrome. True or false?

In recent years the name of Brown-Séquard has been assigned to syndromes caused by spinal cord damage differing widely in extension. On the basis of a review of more than 600 published cases, we propose that Brown-Séquard-plus syndromes be distinguished from the pure Brown-Séquard syndrome. This distinction is shown to be justified on clinical, anatomic, and historical grounds. Two case histories are reported herein to illustrate the essential differences between these syndromes.

Aged↗
PubMed1986

Transient global amnesia after coronary angiography.

A 69-year-old woman suffered transient global amnesia as a complication following coronary angiography. Three reports have been described earlier. The etiology of this condition is discussed. Possibly, this complication is overlooked in some cases.

Aged↗
PubMed1986

Aware or non-aware. On the significance of awareness for the localization of the lesion responsible for homonymous hemianopia.

Some of the patients with homonymous hemianopia are unaware of any visual defect, whereas others perceive this defect more of less consciously. Analysis of the CT scans of 41 cases showed that patients who are partially or fully aware of the hemianopia have purely occipital lesions. The patients who were not aware of the visual defect were found to have larger and more anteriorly situated lesions. Parietal lesions or lesions interrupting the associative pathways to the primary or secondary visual association cortex are considered to be responsible for the lack of awareness of the defect.

Awareness↗
PubMed1986

Bilateral horizontal gaze paralysis due to pontine hemorrhage. A case report.

In a case of bilateral horizontal gaze paralysis, vertical gaze was clinically intact but eye movement recordings demonstrated a transient reduction of vertical saccadic velocities. Horizontal caloric vestibulo-ocular responses were absent. CT scanning and NMR imaging showed a hematoma in the median pontine tegmentum. This case provides additional evidence that vertical and horizontal saccade genesis may be independent of caudal paramedian pontine reticular formation lesions.

Aged↗