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Biomedical subjects

P Jipp

Publications and source records attributed to P Jipp.

At least 37 records · Page 2Linked to original sources
PubMed1984

[Polymyalgia rheumatica and giant cell arteritis].

Among 47 patients with polymyalgia rheumatica there were 30 (63.8%) with temporal giant-cell arteritis. Rapidly progressing visual reduction occurred in 2, acute blindness in 10. Four forms of polymyalgia rheumatica were identified by their different clinical courses, with differing risk of blindness. Those patients with polymyalgia rheumatica and giant-cell arteritis are most endangered who have headaches and neck and shoulder pains, but no or only mild general symptoms.

Aged↗
PubMed1977

[Plasma lipid concentrations during the healing phase after myocardial infarction (author's transl)].

In the initial phase after myocardial infarction the cholesterol and triglyceride concentrations were statistically significantly decreased with respect to the first evaluations. Subsequent analyses during the following four to five weeks revealed some increase in the plasma lipid level. On discharge from hospital the mean plasma lipid concentrations were elevated compared to the first day after myocardial infarction. The time courses of the changes in concentrations of the particular lipids were different. On discharge three quarters of the patients with myocardial infarction had developed hypertriglyceridaemia (hyperlipoproteinaemia type IV or type IIb). Our investigations demonstrate the difficulties in discovering a possible disorder in lipid metabolism during the healing phase after a myocardial infarction.

Cholesterol↗
PubMed1977

Plasma and platelet phospholipids in normal subjects and in patients with chronic arterial obliterations.

Normally a substantially higher percentage of phosphoethanolamine is found in thrombocytes (24%) than in plasma (2.7%). The thrombocytes of patients with chronic arterial occlusions contain significantly less phosphoethanolamine (16.1%) than thrombocytes of normal subjects (24.0%; p less than 0.01). On the contrary, the plasma phosphoethanolamine levels of these patients are significantly elevated (4.8%) in comparison to normal subjects (2.7; p less than 0.05), possibly as the result of some 'leakage' of phosphoethanolamine from platelets. An anti-aggregating treatment did not change the phospholipid pattern of the plasma and of the thrombocytes.

Arterial Occlusive Diseases↗