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P Treit

Publications and source records attributed to P Treit.

6 recordsLinked to original sources
PubMed1986

Free histones in the cells of the rat uterus after neonatal treatment with diethylstilbestrol or allylestrenol.

Neonatal diethylstilbestrol and allylestrenol treatments resulted in a prompt release of free histones that could be demonstrated histochemically by the phosphomolybdic-acid-benzidine reaction. This effect could not be observed in the uterus of 6-10-week old rats but reappeared between 11-14 weeks in the myometrium and uterine epithelium without any repeated treatment. This may be due to the increased production of endogenous steroids. The finding suggest caution when applying steroids to pregnants and neonates.

Allylestrenol↗
PubMed1984

[Obstetrical experiences with adolescents].

Course and results of pregnancies of 661 adolescent girls below 18 years are compared with those of 18 037 controls. Perinatal mortality is twice in the adolescent group than in controls. Toxemias of pregnancy, insufficient labor and intrauterine hypoxia are represented more frequently in adolescent girls.

Adolescent↗
PubMed1980

[Detection of corpus luteum insufficiency by histological study of the endometrium].

Characteristic morphologic alterations of endometrium following corpus luteum insufficiency are described. It is emphasized that in the majority of cases insufficiency of corpus luteum--which in 22 per cent of cases lies on the basis of functional sterility--brings about decreased output of progesterone in the luteal phase of the cycle and induces "disturbed secretion" i. e. hypoluteal transformation of endometrium. Another variant may also take place: normal function of the corpus luteum may last over a too short period inducing the shortening of the secretory phase and the early breakdown of the endometrium. Both, above mentioned mechanisms may inhibit the implantation of the fertilized ovum and lead to functional sterility.

Corpus Luteum↗
PubMed1978

[Ultrastructural changes of the terminal placental villi in placental insufficiency].

Ultrastructural changes of the placenta in toxemia of pregnancy and in diabetes are described. In toxemia of pregnancy in the placenta definite morphological changes were observed; marked atrophy of the syntitial layer was followed by hyperplasia of the cytotrophoblastie cells. The most expressed changes occurred in the vessel wall: swelling of the endothelium, narrowing of the vessel-lumen, deposition of filamentous material in the cytoplasma. Morphological changes in the placenta in diabetes are moderate as compared to those observed in toxemia. Thickening of the basal membrane seems to be most characteristic. Ultrastructural changes are almost identical to those observed in toxemia. Morphological changes revealed may be responsible for the insuffiziency of the placenta and the intrauterine damage to the foetus.

Chorionic Villi↗
PubMed1977

[Ultrastructural changes in the terminal villi of the placenta in placental insufficiency].

Ultrastructural changes of the placenta in toxemia of pregnancy and in diabetes are described. In toxemia of pregnancy in the placenta definite morphological changes were observed; marked atrophy of the syntitial layer was followed by hyperplasia of the cytotrophoblastie cells. The most expressed changes occurred in the vessel wall: swelling of the endothelium, narrowing of the vessel-lumen, deposition of filamentous material in the cytoplasma. Morphological changes in the placenta in diabetes are moderate as compared to those observed in toxemia. Thickening of the basal membrane seems to be most characteristic. Ultrastructural changes are almost identical to those observed in toxemia. Morphological changes revealed may be responsible for the insufficiency of the placenta and the intrauterine damage to the foetus.

Female↗