Pamela Nerheim | PubMed Health

PubMed2003

Irregular atrial activation during atrioventricular nodal reentrant tachycardia: evidence of an upper common pathway.

Controversy continues regarding the precise nature of the reentrant circuit of AV nodal reentrant tachycardia, especially the existence of an upper common pathway. In this case report, we show that marked variation and irregularity in atrial activation (maximum AA interval variation of 80 msec) can exist with fixed and constant activation of the His bundle and ventricles during AV nodal reentrant tachycardia in a 45-year-old female patient. We propose that irregular atrial activation is due to variable and inconsistent conduction from the AV node to the atria through the perinodal transitional cell envelope extrinsic to the reentrant circuit. Our observations support the concept of an upper common pathway, at least in some patients with AV nodal reentrant tachycardia.

Cardiotonic Agents↗

PubMed2001

Syncope.

Successful treatment of patients with syncope depends on the accuracy of the diagnosis, because syncope is a symptom, not a disease; diagnosis of the cause for syncope therefore creates a blueprint for treatment. Most experienced practitioners can diagnose the cause of syncope for less than half of their patients. Excessive and repeat testing is expensive and may not improve the chance of a correct diagnosis. Patient history is the key to the diagnosis. Treatment may vary from a lifestyle change to open heart surgery. The great challenge of treating patients with syncope is to provide cost-effective, safe therapy to those with a benign course and still provide needed treatment for those whose syncope is life threatening.

Journal Article↗