Biomedical subjects

Phil Reed

Publications and source records attributed to Phil Reed.

4 recordsLinked to original sources

PubMed2003

Rats' performance on variable-interval schedules with a linear feedback loop between response rate and reinforcement rate.

Three experiments investigated whether rats are sensitive to the molar properties of a variable-interval (VI) schedule with a positive relation between response rate and reinforcement rate (i.e., a VI+ schedule). In Experiment 1, rats responded faster on a variable ratio (VR) schedule than on a VI+ schedule with an equivalent feedback function. Reinforced interresponse times (IRTs) were shorter on the VR as compared to the VI+ schedule. In Experiments 2 and 3, there was no systematic difference in response rates maintained by a VI+ schedule and a VI schedule yoked in terms of reinforcement rate. This was found both when the yoking procedure was between-subject (Experiment 2) and within-subject (Experiment 3). Mean reinforced IRTs were similar on both the VI+ and yoked VI schedules, but these values were more variable on the VI+ schedule. These results provided no evidence that rats are sensitive to the feedback function relating response rate to reinforcement rate on a VI+ schedule.

Animals↗

PubMed2003

The effect of signaled reinforcement on rats' fixed-interval responding.

Four experiments examined the effect on rats' response rate of presenting a brief (500 ms) stimulus simultaneously with the delivery of food on fixed-interval (FI) schedules. In Experiment 1, reinforcement signals that were spatially diffuse (both tones and lights) elevated rates of responding, but responding was attenuated by localized visual stimuli. The remaining experiments examined the signal-induced potentiation of responding. In Experiment 2, a tone reinforcement signal potentiated response rates on an FI schedule, but attenuated response rates on a variable-interval (VI) schedule. This difference was obtained even though the overall rate of responding was equated on the two schedules before the introduction of the signal. Signal-induced potentiation of responding occurred over a range of FI values employed in Experiment 3. In Experiment 4, presenting a reinforcement signal when high local rates of response had occurred immediately before reinforcement resulted in potentiated rates of responding on an FI schedule. The opposite effect on response rate occurred when the reinforcement signal followed only low local rates of response. These results indicate that a variety of factors influence the effects of a reinforcement signal. They imply, however, that the local rate of response at the time of reinforcement is a key factor in establishing the nature of the signaling effect.

Animals↗

PubMed2003

Human causality judgments and response rates on DRL and DRH schedules of reinforcement.

The effect of various relationships between a response (an investment made in the context of a game) and an outcome (a return on the investment) on judgments of the causal effectiveness of the response was examined. In Experiment 1, response rates and causal judgments were higher for a differential-reinforcement-of-high-rate (DRH) schedule relative to a variable-ratio (VR) schedule with the same probability of outcome following a response. Response rates were also higher for a DRH than for a variable-interval schedule matched for reinforcement rate. In Experiment 2, response rates and causal judgments were lower for a differential-reinforcement-of-low-rate schedule relative to a VR schedule with the same probability of outcome following a response. These results corroborate the view that schedules are a determinant of both response rates and causal judgments, and that few current theories of causal judgment explicitly predict this pattern of results.

Adolescent↗

PubMed2002

The influence of a distractor during compound preexposure on latent inhibition.

Nonreinforced exposure to a nontarget stimulus that was followed by nonreinforced exposure to a target/nontarget simultaneous compound stimulus resulted in enhanced latent inhibition of the target. Conditioning was slower after this treatment than after nonreinforced exposure to the target stimulus alone (Experiment 1). However, a salient auditory stimulus presented immediately after the compound in the second phase reduced levels of latent inhibition, relative to the enhanced latent inhibition produced when no such extracompound stimulus was presented (Experiments 2 and 3). This effect was not noted if the salient auditory cue was presented 10 sec after the termination of the compound stimulus (Experiment 4). In Experiment 5, there was no disruption of simple latent inhibition produced by a salient stimulus. These results are consistent with enhanced latent inhibition's being produced by the formation of within-compound associations, which are disrupted by the salient extracompound stimuli.

Animals↗