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Biomedical subjects

R Bouchard

Publications and source records attributed to R Bouchard.

At least 55 records · Page 3Linked to original sources
PubMed1979

[Radiological study of 305 cases of spondylolysis with or without spondylolisthesis].

Three hundred and five cases of spondylolysis with or without spondylolisthesis were reviewed. The concept of an acquired lesion must be retained even though a dysplastic lesion may be postulated to account for the familial incidence. The examination in erect position facilitates the diagnosis by favoring slipping. The vast majority of lesions are found at L5-S1. Spina bifida occulta is often found in association with spondylolisthesis. This lesion leads to apophyseal arthrosis and disc degeneration at the involved level. The accentuation of the lombosacral angle is not a predominent factor leading to spondylolysis. This angulation will increase with duration and degree of slipping. CT scan provides an excellent account of the state of the spinal canal at the affected level. Could pseudospondylolisthesis be a headed stage of spondylolysis?

Adolescent↗
PubMed1979

Ewing's sarcoma in adults.

Twenty-two adult patients, aged 18 and over, with Ewing's sarcoma of bone, were evaluated clinically and radiographically. They had clinical findings similar to younger patients with Ewing's sarcoma, except that they presented with fewer systemic symptoms. The radiographic characteristics included a preponderance of flat bone involvement (54%) over long bone involvement (36%). A soft-tissue mass was detected in only 55% of the cases. CT scans, when used, proved to be helpful in delineating a soft-tissue mass associated with the bony lesion, and in follow-up after therapy.

Adolescent↗
PubMed1978

Autosomal recessive spastic ataxia of Charlevoix-Saguenay.

A new syndrome of autosomal recessive spastic ataxia has been isolated in the Charlevoix-Saguenay region of Quebec. This syndrome is remarkably homogeneous and includes: spasticity, dysarthria, distal muscle wasting, foot deformities, truncal ataxia, absence of sensory evoked potentials in the lower limbs, retinal striation reminiscent of early Leber's atrophy and the frequent presence (57%) of a prolapse of the mitral valve. Biochemically, many cases show impaired pyruvate oxidation, others have hyperbilirubinaemia and some have low serum beta-lipoproteins and HDL apoproteins. These features are similar to those found in typical Friedreich's ataxia.

Adolescent↗