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Biomedical subjects

R Chand

Publications and source records attributed to R Chand.

10 recordsLinked to original sources
PubMed1995

Xanthomonas campestris pv. parthenii pathovar nov. incitant of leaf blight of parthenium.

A new bacterial leaf blight disease of parthenium (Parthenium hysterophorus L.) is described for the first time. The disease-causing bacterium was isolated and its morphological, physiological and biochemical characters were determined. The pathogenicity of bacterium is apparently limited only to parthenium. The pathogen was identified as Xanthomonas campestris pv. parthenii pathovar nov. on the basis of morphological, physiological, biochemical and pathogenic characteristics.

Energy Metabolism↗
PubMed1995

Pancreatic enzyme supplementation in acute pancreatitis.

This study evaluates the effect of oral pancreatic enzyme supplements on pain, analgesic requirement and the incidence of complications in patients with acute pancreatitis. This double blind, prospectively randomised placebo controlled study included 23 patients. Pain was monitored using a visual analogue scale; the analgesic requirement was assessed with a numerical score. No significant differences were noted between the median (range) pain scores of patients who received placebo: 22 (17.1-58) and those who received enzymes: 23 (11.3-63). Hospital stay was 7 (5-10) days in patients on placebo and 8 (6-24) days in the enzyme group (p = 0.069). Analgesic requirements were: placebo 20 (6-60) and enzymes: 16 (0-63) (p = 0.56). This study has shown no beneficial effect of oral pancreatic enzyme supplements in the initial management of patients with acute pancreatitis.

Acute Disease↗
PubMed1986

Mercuric chloride-induced physiological dysfunction in Notopterus notopterus.

The freshwater fish Notopterus notopterus were exposed to various sublethal concentrations (0.088, 0.044, 0.029, 0.022, and 0.017) of mercuric chloride (HgCl2) mg/liter for 15, 30, 45, and 60 days, and their effects on glucose, lactate, cholesterol, plasma glutamic oxalacetic transaminase (PGOT) and plasma glutamic pyruvic transaminase (PGPT) of the blood were observed. Lactate increased significantly (P less than 0.05; P less than 0.01; P less than 0.001) and glucose. PGOT, and PGPT increased significantly (P less than 0.05; P less than 0.01). However, cholesterol decreased significantly (P less than 0.05; P less than 0.01; P less than 0.001) in the blood of N. notopterus.

Alanine Transaminase↗
PubMed1985

In vivo accumulation and effects of mercuric chloride on tissue phosphatases of Notopterus notopterus.

Sublethal concentrations of mercuric chloride inhibited the liver, kidney and gill enzymes, acid, alkaline, and glucose-6-phosphatase of the fish Notopterus notopterus, which had been exposed to the heavy metal for 30 days. Higher concentrations produced highly significant (p less than 0.001) inhibition, whereas concentrations that were 1/25th the lethal concentration caused significant (p less than 0.05)/insignificant inhibition. The quantity of mercuric chloride accumulated in tissues correlated with the degree of enzyme inhibition.

Animals↗
PubMed1984

Mercuric chloride stress on serum transaminase activity in Notopterus notopterus.

Mercuric intoxication at sublethal levels (1/5, 1/10, 1/15, 1/20 or 1/25 fractions of 96 h LC50) produced alterations in the activity of serum glutamic oxalacetic transaminase (SGOT) and serum glutamic pyruvic transaminase (SGPT) of Notopterus notopterus. The difference between control and treated fish was found to be significant at P less than 0.05, P less than 0.01 and P less than 0.001 levels. Maximum (81.75%) significant (P less than 0.001) effect was observed in SGOT: maximum (69.23%) significant (P less than 0.001) effect was observed in SGPT in fish exposed for 60 days.

Alanine Transaminase↗