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Biomedical subjects

R D Stewart

Publications and source records attributed to R D Stewart.

At least 163 records · Page 9Linked to original sources
PubMed1974

Correlation between long-acting thyroid stimulator protector level and thyroid 131-I uptake in thyrotoxicosis.

Out of 50 consecutive untreated patients with diffuse toxic goitre 15 showed long-acting thyroid stimulator (LATS), 30 showed LATS protector only, and five showed neither. LATS protector was present in all the patients with LATS. Infiltrative ophthalmopathy was less common in patients with LATS protector only (40%) than in patients with LATS also (67%), but the difference was not significant. There was a correlation between LATS protector level and thyroid (131)I uptake rate factor (k(1)), the coefficient (r) being 0.68 (P < 0.001). LATS level showed no such correlation. The results support the hypothesis that LATS protector is a pathogenic thyroid stimulator in patients with diffuse toxic goitre.

Animals↗
PubMed1974

"Degreaser's flush".

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Atmosphere Exposure Chambers↗
PubMed1973

Carboxyhemoglobin concentrations in blood from donors in Chicago, Milwaukee, New York, and Los Angeles.

To determine the carbon monoxide exposure experienced by the residents of Chicago, Los Angeles, Milwaukee, and New York, venous blood samples were obtained from adults at arbitrarily chosen blood bank collection sites in the four cities and analyzed for circulating carbon monoxide, carboxyhemoglobin. For comparative purposes, blood was obtained from volunteers breathing carbon monoxide-free air and was found to contain 0.45 percent carboxyhemoglobin. By contrast a high percentage of all the nonsmoking blood donors breathing city air had carboxyhemoglobin saturations greater than 1.5 percent, which indicated that exposure to carbon monoxide in excess of that permitted by the quality standards of the Clean Air Act of 1971 was widespread and occurring regularly.

Air Pollution↗
PubMed1972

Carboxyhemoglobin elevation after exposure to dichloromethane.

Inhalation of dichloromethane vapor in concentrations of 500 to 1000 parts per million for 1 to 2 hours promptly initiated the formation of significant quantities of carbon monoxide in human subjects. The evidence suggests that carbon monoxide may be a metabolite of dichloromethane and, that exposure to concentrations of dichloromethane below the industrial threshold limit values may result in the formation of carbon monoxide in amounts that exceed the allowable limits.

Adult↗