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Biomedical subjects

R K Stroud

Publications and source records attributed to R K Stroud.

21 records · Page 2Linked to original sources

Isolation of Edwardsiella tarda from three Oregon sea mammals.

Edwardsiella tarda was isolated from the peritoneal exudate of a Steller's sea lion (Eumetopias jubata) with peritonitis resulting from a perforating colonic ulcer; from the liver of a harbor porpoise (Phocena phocena) with metritis and peritonitis sequela to dystocia; and from the liver of a California sea lion (Zalophus californianus) with peritonitis following fracture and necrosis of a lumbar vertebrae. These findings indicate E. tarda is a common opportunistic invader in sick or injured marine mammals.

Animals↗

Bovine anaplasmosis transmission studies conducted under controlled natural exposure in a Dermacentor andersoni = (venustus) indigenous area of eastern Oregon.

In Anaplasma marginale transmission studies conducted on the high semi-arid range of eastern Oregon during the 1974 and 1975 vector season, A marginale-susceptible calves (principals) were maintained on 2 raised tick-proof platforms. Anaplasmosis-susceptible control calves of approximately the same age and latent-infected cows grazed the area surrounding the platforms. One latent-infected steer spent the entire 1975 vector season on a platform with the principals. The 28 principals did not develop anaplasmosis, whereas 15 of 30 (50%) controls became infected. The disease was not transmitted from the latent-infected cattle to the principals exposed only to flying hematophagous insects, whereas 50% of the controls exposed to the Rocky Mountain wood tick Dermacentor andersoni) = (venustus) developed the disease. Dermacentor andersoni appears to be the principal vector on this range.

Anaplasmosis↗

Suspected fusariomycotoxicosis in sandhill cranes (Grus canadensis): clinical and pathological findings.

In 1985 and 1986, large-scale natural die-offs of sandhill cranes in Texas were attributed to fusariomycotoxicosis. These birds demonstrated a progressive loss of motor control to the neck, wings, and legs. Based on necropsy and/or histopathology of 31 cranes, the most common lesions involved skeletal muscle and included hemorrhages, granulomatous myositis, thrombosis, and vascular degeneration. Serum chemistry results revealed that levels of creatinine kinase, aspartate aminotransferase, and alanine aminotransferase were above published normals. However, only alanine aminotransferase was higher in clinically affected cranes than in normal cranes collected from the same area.

Alanine Transaminase↗