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R Saison

Publications and source records attributed to R Saison.

15 recordsLinked to original sources
PubMed1980

Canine blood groups. 1. Description of new erythrocyte specificities.

Methods of production and characterization of immune typing reagents reactive with the erythrocytes of the dog are described. Five of the reagents (agglutinins) were reactive with previously unknown blood group factors and were shown by statistical and genetic tests on 222 random samples and 28 families to belong to 5 previously unrecognized blood group systems. The five new systems were one-blood factor, two-allele, three-genotype systems like the B, C, D and F systems. These were arbitrarily named J, K, L, M and N.

Animals↗
PubMed1980

Canine blood groups. 2. Description of a new allele in the Tr blood group system.

A previously unrecognized canine red cell antigen, tentatively named O, was found after absorption analysis of an alloimmune antiserum and absorption of several immune or naturally occurring cross-reacting heteroantibodies from man, cattle and swine. The serological results and genetic analysis of a limited number of complete dog families indicated that the new factor probably belongs to the Tr blood group system. No individual possessed both factors Tr and O, and a large proportion of animals was negative for both factors. The serological pattern for the new Tr system obtained was consistent with similar systems observed in sheep (A-O), pig (R-O) and man (Bombay) in which the gene for one factor was dominant over and masked the gene for the second factor in the system. The negative phenotype was accounted for by the actions of an epistatic gene.

Alleles↗
PubMed1966

Viral plasmacytosis (Aleutian disease) in mink. VI. The development of positive Coombs test in experimental infections.

Coombs tests became positive coincident with the development of plasmacytosis in experimentally infected mink. These observations are discussed as possible evidence of an auto-immune phenomenon. Several mechanisms are suggested in which a virus as primary etiologic agent might promote the observed plasmacytosis, hypergammaglobulinemia, and erythrocyte sensitization.

Animals↗