[Psoas abscess].
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Diabetes Mellitus, Type 2↗
Biomedical subjects
Raúl Carrillo-Esper
Publications and source records attributed to Raúl Carrillo-Esper.
[Persistent left superior vena cava: infrequent localization of central venous catheter].
OBJECTIVE: Our objective was to report a Persistent Left Superior Vena Cava in a critically ill patient. DESIGN: Case report. PLACE: Intensive care unit of a referral center. PATIENT: A 52 years-old male without previous cardiovascular disease was admitted to the intensive care unit due to cranial trauma. A central left subclavian vein catheter was placed at his arrival at the center chest X-ray showed the catheter in the left of the cardiac silhohuete without hemo-pneumothorax. A angiographic evaluation through central venous catheter showed persistent left superior vena cava draining to the coronary sinus and right atrium with normal permeability of right superior vena cava. Other congenital alterations were excluded. Chest computed tomography confirmed the diagnosis. Persistent left superior vena cava is a congenital vascular abnormality. The intensive care unit staff must be aware of this vascular abnormality because it can complicate central venous catheterization.
Angiography↗
[Knotting Swan-Ganz catheter in right pulmonary artery].
Knotting Swan-Ganz catheter is a rare complication of hemodynamic monitoring. This is the second case reported in Mexico in over 20 years. Identified risk factors that increase likelihood of loop formation knotting of the catheter are blind introduction and small diameter of catheter, incomplete balloon inflation before advancing it, several tries to wedge it, bending of the catheter by heat, and enlarged right heart chambers. More frequent sites of knotting are right atrium and ventricle, subclavian vein, and pulmonary artery and its branches. Diagnostic confirmation is made by chest radiography, fluoroscopy, and echocardiography. Therapeutic approach depends on knot characteristics, either non-surgical or surgical.
Catheterization, Swan-Ganz↗
[Innate immunity, Toll receptor and sepsis].
The innate immune response is the first line of defense against infection. Toll-like receptors (TLRs) recognize bacterial lipopolysaccharide and other pathogen-associated molecular patterns (PAMPs). Intracellular signals initiated by interaction between Toll receptors and specific PAMPs results in inflammatory response. Sepsis and septic shock are the result of an exaggerated inflammatory systemic response induced by innate immune dysregulation.
Animals↗
[Vasopressin use in shock].
Arginine-vasopressin (VP), also known as the antidiuretic hormone, is essential for water homeostasis. Its synthesis and liberation depends on regulation of osmotic, hypovolemic, hormonal, and nonosmotic stimuli. It has been demonstrated that it is key for maintenance of cardiovascular homeostasis through vasomotor regulation, the determinant of systemic vascular resistance and mean arterial pressure, a process acting through V1 receptors. Shock state with refractory vasodilation seen in sepsis, systemic inflammatory response, hypovolemia, cardiac arrest, polytrauma, etc., is characterized by an initial phase of liberation and increased levels of VP followed by a second phase characterized by inappropriately low levels of this hormone that are associated with refractoriness to management with volume, inotropics, and vasopressors. It has been demonstrated in clinical and experimental studies that exogenous VP treatment under this condition increases systemic vascular resistance, perfusion pressure, and oxygen supply to peripheral tissues, which makes it possible to decrease and to suspend vasopressors and also to increase survival.
Clinical Trials as Topic↗