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Biomedical subjects

S A Howlett

Publications and source records attributed to S A Howlett.

9 recordsLinked to original sources
PubMed1976

Eosinophilic gastroenteritis.

Four patients with eosinophilic gastroenteritis are presented to illustrate the protean manifestations of this disorder. While the cause is unknown, the complications many, and the differential diagnosis often challenging, the treatment is simple and consists of low-dose, alternate-day administration of corticosteroids. Diagnosis depends on gastrointestinal biopsy, which usually can be obtained without surgery.

Biopsy↗
PubMed1975

Cholangitis complicating congenital hepatic fibrosis.

A patient with cholangitis complicating congenital hepatic fibrosis is presented. In contrast to most other reported cases, many of whom have been treated surgically, this patient had a benign course. The likelihood that this complication of congenital hepatic fibrosis will respond to appropriate conservative therapy suggests that more aggressive forms of treatment should be reserved for those patients who fail to respond to medical management.

Biopsy, Needle↗
PubMed1975

Renal failure associated with acetazolamide therapy for glaucoma.

Presented is a case of acute renal failure induced by acetazolamide therapy for glaucoma. The 69-year-old patients recovered rapidly after oral administration of fluids and sodium bicarbonate. This sulfonamide like nephropathy should be differentiated from acetazolamide-related calcium phosphate nephrolithiasis.

Acetazolamide↗
PubMed1975

Perforation of duodenal stump ten years after partial gastrectomy.

Described is a patient who developed spontaneous perforation of the duodenal stump of a Billroth II anastomosis done ten years ago. This catastrophe is thought to have resulted from acute afferent-loop obstruction, a rare but recognized late postgastrectomy complication. The patient died of hemodialysis disequilibrium syndrome.

Adult↗
PubMed1975

Inhibition of macrophage migration in response to hepatitis Bs antigen.

Macrophage migration inhibition factor produced in response to purified hepatitis Bs antigen has been demonstrated in patients with and recovering from hepatitis B antigen-positive acute viral hepatitis. These results suggest that cell-mediated immunity against this antigen occurs concomitantly with the illness, and may be important in its pathogenesis. In addition, patients with chronic aggressive hepatitis, whether serologically positive or negative for hepatitis Bs antigen, were also found to have positive macrophage migration inhibition tests. This observation suggests a role for hepatitis B virus in the majority of patients with chronic aggressive hepatitis. Negative macrophage migration inhibition tests in response to the antigen were noted in acute viral hepatitis seronegative for the hepatitis B antigen, alcoholic hepatitis, normal controls, and carriers of the antigen.

Adult↗