[Particular features of Helicobacter pylori contamination of the stomach mucous coat in patients with different types of the chronic gastritis].
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Biomedical subjects
Publications and source records attributed to S A Povzun.
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In patient who died from anaphylactic shock the level of immunoglobulins E in postmortem blood exceeded the upper limit almost 6 times this allowing to confirm the diagnosis. It is stated that a postmortem blood examination may be useful in the autopsy diagnosis of anaphylactic shock.
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89 liver needle biopsies from young persons--HBV and HCV carriers--were studied histologically, immunohistochemically, morphometrically and electron microscopically. Groups with compensatory-adaptive and degenerative liver lesions, those with mild manifestations of a chronic virus infection, and those with chronic hepatitis of minimal and weak activity were identified. Latent chronic hepatitis is found in 68.5% of HBV and HCV carriers. Morphological picture of HCV infection is more pronounced as compared to that of HBV infection. The role of eosinophils in the development of hepatitis B and C forms lacking clinical symptoms is demonstrated.
In 1930-1940 the pathology chair of the Military Medical Academy participated in organization of military pathological anatomy service. The activity of the chair staff goes in two directions: management of pathology service activity in the army both at peace and war time and research in combat pathology.
The following methods of microscopic diagnosis training are proposed: the analysis of microscopic slides using the microscope equipped with video camera visualizing images on the screen of telemonitor with a group of 2-4 physicians; situation tasks using histological slides from legal postmortems; discussion with trainees of their conclusions on current biopsies obtained by these trainees; situational tasks on the sets of biopsies of last years.
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An analysis of 50 case histories of surgical patients who died from nonhepatic diseases has shown that acute hepatic insufficiency is often hyperdiagnosed. Possible causes of this hyperdiagnosis resulting from insufficient information and wrong interpretation of laboratory findings are discussed.