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S Arvan

Publications and source records attributed to S Arvan.

27 records · Page 2Linked to original sources
PubMed1984

Mural thrombi in coronary artery disease. Recent advances in pathogenesis, diagnosis, and approaches to treatment.

Detection of ventricular mural thrombi has been facilitated by the introduction of echocardiography, nuclear angiography, and radioactive labeling of platelets. Most of the data regarding the incidence of mural thrombi, pathogenesis of thrombus formation, and predisposing factors for systemic emboli after myocardial infarction that were reported on postmortem studies in the past can now be reexamined in the living patient with these noninvasive methods. The data accumulated from such investigations are now available and the results are reviewed herein. A critique of the usefulness of echocardiography and nuclear methods is also presented.

Angiography↗
PubMed1983

Pericardial tamponade in a patient with treated myxedema.

Pericardial tamponade developed in a patient with hypothyroidism and a pericardial effusion after she had been successfully treated with levothyroxine sodium. Refractory pericardial effusion is an unusual complication of treated myxedema. A possible mechanism for its occurrence is presented.

Aged↗
PubMed1982

M-mode and two-dimensional echocardiography of pulmonary emboli.

The transit of peripheral emboli on two-dimensional echocardiography through the right side of the heart has not been reported before, to the best of our knowledge. Although this finding was fortuitous and unexpected in view of the transient and unpredictable nature of systemic venous emboli, it was a clinically important finding since it alerted the physician and ultimately changed the therapeutic approach. We suggest that echocardiography can detect impending pulmonary emboli by identifying random, bizarre signals reflected from the fibrin particles in the right atrium and right ventricle. Other conditions, as already mentioned, must first be excluded in order to arrive at the correct diagnosis.

Aged↗
PubMed1982

Embolization of a left ventricular mural thrombus: verification by two-dimensional echocardiography.

A two-dimensional (2-D) echocardiogram in a 69-year-old man disclosed a septal-apical left ventricular thrombus six days after an acute anterior wall myocardial infarction. On the ninth hospital day, he suddenly experienced left-sided weakness and a left-sided field cut consistent with the diagnosis of a cerebrovascular accident (CVA). A repeated 2-D echocardiogram showed that the original mural thrombus had completely disappeared, which strongly supported an embolic event as the cause of the CVA. Two-dimensional echocardiography helped to verify the cause of this patient's neurologic problem. The mechanism and predisposing factors that led to a systemic embolism are discussed.

Aged↗
PubMed1977

Effects of actodigin on the heart.

Actodigin is a new semisynthetic cardiac glycoside reported to have a rapid onset and brief duration of action in dogs. Five patients with congestive heart failure in normal sinus rhythm were given incremental doses of actodigin. Overall, there was no significant change in heart rate, aortic or pulmonary artery pressure, systemic vascular resistance, cardiac index, and stroke volume. This lack of response to actodigin is consistent with previous reports of acute administration of other cardiac glycosides. Four patients with atrial fibrillation and a rapid ventricular rate were given similar doses of actodigin. The ventricular rate was readily controlled. After drug administration was stopped, the ventricular rate quickly returned toward predrug levels. Thus, the rapid onset and brief duration of action of actodigin may be useful in the initial management of atrial fibrillation.

Adult↗
PubMed1976

Echocardiographic findings in a patient with Candida endocarditis of the aortic valve.

The echocardiographic appearance of fungal endocarditis of the aortic valve is described in a patient who subsequently died from this disease. In addition, the progressive growth of the vegetation on serial echocardiograms was recorded, and premature closure of the mitral valve was absent, notwithstanding perforation of two aortic cusps.

Aortic Valve↗
PubMedDate not supplied

Left ventricular mural thrombi secondary to acute myocardial infarction: predisposing factors and embolic phenomenon.

Twenty-five patients with acute myocardial infarction were examined for ventricular thrombi using two-dimensional echocardiography. Six of 10 patients (60%) with an anterior wall infarction had an apical or apical-septal thrombus within the first week of hospitalization. None of the fifteen patients with an inferior wall myocardial infarction had a mural thrombus. Although the size of infarction in the patients with a thrombus was not significantly larger than in those who had an anterior wall infarction without a thrombus (43% +/- 10% vs. 31% +/- 7%, P less than 0.1), the severity and extent of dyskinesia or akinesia were more marked in the former group. Left ventricular function as determined by the nuclear blood pool scan ejection fraction was also significantly less for the former group than for the latter group (21% +/- 6% vs. 40% +/- 11%, P less than 0.02). Three of six patients with an intracavitary thrombus on echocardiography had systemic embolic during their hospital course. Postinfarction ventricular thrombi tend to occur in those patients with an anterior wall myocardial infarction who have far advanced wall motion abnormalities of the affected area, and overall poor left ventricular function. Although the number of patients was small, the high incidence of systemic embolization in the infarction subjects with echocardiographically proven thrombi indicates that these patients are at increased risk for such events.

Aged↗