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Biomedical subjects

S G Genes

Publications and source records attributed to S G Genes.

At least 19 recordsLinked to original sources
PubMed1981

[Diabetic neuropathy and lesion of the vessels].

Data from the literature concerning the frequent lack of correlation between the clinical manifestations of diabetic neuropathy on the one hand and the morphofunctional status of the appropriate nerves, disorders in carbohydrate and lipid metabolism, and the influence of antidiabetic therapy on metabolism and the clinical picture of diabetic neuropathy on the other, are presented. In this connection, abundant evidence of frequent affection of various blood vessels, particularly small and smallest, in diabetic neuropathy is considered. But even these data attest to a non-infrequent lack in diabetic neuropathy of the involvement of the appropriate vessels, particularly the lack of basal membrane thickening which is a most common indication to the diabetic involvement of capillaries. Attention is drawn to the possible role of genetic heterogeneity of people with regard to the development of various forms of diabetic neuropathy.

Adult↗
PubMed1977

[Role of the liver in hormone metabolism and in the regulation of their content in the blood].

The normal liver function depends upon the amount of hormones in the blood which is regulated not only by the endocrine glands (EG) but by the liver itself to a large degree. The hormone content in the blood may be normal, when the EG function is increased, if the liver removes them intensively, inactivates them and excretes in the bile. If the hormone excess exceeds the regulatory capacities of the liver, their level in the blood increases. An inflow of an increased amount of hormones damages liver functions, then elimination of the hormone excess decreased or does not occur, and their level in the blood increases further. The function of the corresponding EG is inhibited. When the EG functions decrease, the level of hormones in the blood may remain normal owing to their decreased removal by the normal liver. Thus, the normally functioning liver may for some time maintain the normal balance of hormones when the EG functions are disturbed. The same may happen in the disorders in the liver function, if the EG function normally.

Adaptation, Physiological↗