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Biomedical subjects

S H Auerbach

Publications and source records attributed to S H Auerbach.

10 recordsLinked to original sources

Pure word deafness. Analysis of a case with bilateral lesions and a defect at the prephonemic level.

We have demonstrated that the deficit in speech perception in our patient with pure word deafness is secondary to a prephonemic temporal auditory acuity disorder. We delineated the nature of the auditory processing deficit in our patient with bilateral lesions and then demonstrated the presence of a predicted deficit in phonemic discrimination. This pattern is comparable to previous cases with bilateral lesions and distinct from other cases with unilateral lesions. Review of previous reports suggests that there are two distinct types of pure word deafness: type 1, in which the deficit is prephonemic and related to a temporal auditory acuity disorder, and type 2, a form that is independent of a temporal auditory acuity disorder, and has a deficit in linguistic discrimination that does not adhere to a prephonemic pattern (Denes and Semenza, 1975; Saffran et al., 1976). The former has been associated with bilateral temporal lobe lesions (Naeser, 1974; Chocholle et al., 1975), the latter with left unilateral lesions (Denes and Semenza, 1975; Saffran et al., 1976). The first form is an apperceptive disorder, whereas the second represents a higher disorder in phonemic discrimination and may be considered a fragment of Wernicke's aphasia.

Agnosia

Familial recurrent peripheral facial palsy. Observations of the pediatric population.

Idiopathic peripheral facial palsies in the pediatric population have been noted to carry a favorable prognosis. We describe three members of a family, including two children with recurrent facial palsy, and review the relevant literature. As recurrent peripheral facial palsies carry a relatively poor prognosis, the physician should be alerted to this possibility in any child who has an idiopathic peripheral facial palsy and a positive family history.

Adolescent

Headache in cerebral embolic disease.

A patient is presented who had severe headache and then developed a stuttering course of clinical neurological deficits. Arteriography demonstrated an ipsilateral proximal middle cerebral embolus. With recurrence of symptoms, repeat arteriography showed another, more proximal embolus with areas of distal occlusion. The headache probably occurred when the embolus became lodged in a pain sensitive cerebral vessel at the base of the brain. The subsequent stuttering neurological deficits were due to fragmentation of the embolus as well as recurrence.

Cerebral Angiography

Pharmacologic treatment of cognitive deficits: a case study.

Traumatic brain injury (TBI) is associated with a variety of cognitive and behavioural disorders. A few reports have suggested that pharmacotherapy might be useful in the management of these disorders. We present a case of a patient who suffered a left subdural hematoma and herniated. Several months after the injury, his function was severely limited. Nevertheless, he demonstrated a clear improvement in response to oral methylphenidate and parenteral physostigmine. We conclude that specific pharmacotherapy may result in major cognitive and functional improvements in severely impaired patients.

Adult