Changes induced by manganese in fish testis.
Exposure of Colisa fasciatus, a freshwater teleost, to 2500 mg/l manganese sulphate for 90 h caused decreased spermatogenic activity and hemorrhage in the testes.
Biomedical subjects
Publications and source records attributed to S J Agrawal.
Exposure of Colisa fasciatus, a freshwater teleost, to 2500 mg/l manganese sulphate for 90 h caused decreased spermatogenic activity and hemorrhage in the testes.
Blood dyscrasia in a fish exposed to experimental manganese poisoning is described. Colisa fasciatus, a fresh water teleost, showed significant decreases in the total erythrocyte count, number of erythrocytes/1000 blood cells of all types, and in the hepatosomatic index (relative liver wt) after exposure for 90 h to 2500 mg/l manganese sulphate; the 96 h LC50 value was 2850 mg/l. The exposure also evoked leucocytosis due to an increase in the number of small lymphocytes. Other haematological characteristics, viz., total count of thrombocytes, distribution of immature red blood cells, thrombocytes and large lymphocytes/1000 blood cells, haematocrit, erythrocyte sedimentation rate, haemoglobin, and clotting time, did not differ significantly from the controls.
Explore the source record for details and available documents.
Colisa fasciatus, a fresh water teleost, when exposed for 90 hrs to a sublethal concentration of 195 p.p.m. (0.87 of the 96 hrs LC50 value) cobalt elicited leucopenia due to reduction in the number of circulating small lymphocytes. The treatment also caused decline in blood clotting time with a concomitant increase in the abundance of circulating thrombocytes. No differences in total erythrocyte count, haematocrit, erythrocyte sedimentation rate, and haemoglobin values were found between the control and treated fish. The hepatosomatic index of fish increased significantly after exposure to cobalt. Results are discussed in terms of fish's reaction to acute stress.
Colisa fasciatus, a freshwater teleost, were exposed for 90 hrs to 45 p.p.m. nickel sulphate under static test conditions. The treatment resulted in leucopenia due to reduction in the number of small lymphocytes and polycythemia with concomitant increases in the haematocrit and haemoglobin values, and in retardation of the erythrocyte sedimentation rate of the moribund fish. No differences in total thrombocyte count, clotting time, and hepatosomatic index were found between the control and the treated fish. Lymphopenia and erythrocytosis may be used in presumptive monitoring nickel toxicity in fish.