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Biomedical subjects

S R Minkin

Publications and source records attributed to S R Minkin.

7 recordsLinked to original sources
PubMed1993

[Valvular prolapse (clinical, echocardiographic, phonocardiographic and electrocardiographic features)].

Clinical, echoCG, PCG and ECG examinations were performed in 42 patients with primary isolated valve prolapses: mitral, aortal and tricuspid (32, 5 and 5 patients, respectively). It is shown that degree 1 regurgitation caused no hemodynamic disturbances, that of degree 2 and 3 brought about cardial complaints in more than half the patients, hemodynamic overstrain of the heart, sound symptoms similar to those of relevant valvular disease.

Adolescent↗
PubMed1993

[Atherosclerotic heart defects].

Clinical, echocardiographic, Doppler echocardiographic, electro- and phonocardiographic studies were performed in 31 patients with cardiac atherosclerotic lesions (aortic in 17 patients, aortomitral in 10, mitral in 4) and in 20 patients matched for age, sex, and underlying abnormality, but without cardiac disease. Atherosclerotic defects caused by calcification of the valvular apparatus were shown to be rarely (5.9% of patients) isolated, they were more frequently combined or concurrent. Atherosclerotic defects caused no significant myocardial structural and functional changes in addition to the underlying pathology. Aortic atherosclerotic stenosis is diagnosed at equal rates by using echo and phonocardiography, whereas aortic valvular insufficiency and mitral diseases are better (7- and 9-fold) detected with echocardiography and Doppler echocardiography, whereas aortic valvular insufficiency and mitral diseases are better (7- and 9-fold) detected with echocardiography and Doppler echocardiography than with phonocardiography.

Aortic Valve↗
PubMed1991

[The echocardiographic signs of left ventricular hypertrophy in persons with borderline arterial hypertension and hypertensive disease and their relatives].

Altogether 122 persons underwent echocardiographic studies. All the persons examined were distributed into 5 groups: group 1 comprised healthy persons without hereditary aggravation as regards essential hypertension (EH); group 2 included healthy persons with aggravated heredity as regards EH; group 3 was made up of persons with borderline arterial hypertension; group 4 of persons with labile EH; group 5 of persons with stable EH. Early appearance of the signs of left ventricular hypertrophy was recorded in persons with hereditary aggravation as regards EH. The changes were particularly remarkable in aggravated heredity as regards both parents or mother. The patients with stable EH demonstrated, in addition to an increase of the mass of the left ventricle, enlargement of the cavity of the left atrium and the width of the aorta.

Adult↗