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Biomedical subjects

S S Hamer

Publications and source records attributed to S S Hamer.

8 recordsLinked to original sources
PubMedDate not supplied

Digitalis excess mimicking the sick sinus syndrome.

Symptomatic bradycardia-tachycardia is often a feature of sick sinus syndrome. However, when alternating brady-tachycardia results from excessive use of digitalis preparations or electrolyte imbalance, it is iatrogenic and therefore not considered to be SSS per se. Sick sinus syndrome is applied to the following arrhythmias providing they are not iatrogenically induced: (1) sinus bradycardia usually unresponsive to atropine therapy or exercise, (2) sino-atrial block or arrest with long pauses, and (3) alternating bradycardia or tachycardia.

Arrhythmias, Cardiac↗
PubMedDate not supplied

Pseudo A-V block. Part I.

An example of junctional premature beats with concealed conduction has been presented. The arrhythmia mimics Mobitz Type II second-degree A-V block. Awareness of this arrhythmia when added to the fact that Mobitz Type II block is rarely seen with IWMI will prevent an improper or hasty diagnosis. Cardiac arrhythmias should be interpreted and managed in context of the clinical setting.

Arrhythmias, Cardiac↗
PubMedDate not supplied

Supraventricular tachycardia that mimics ventricular tachycardia.

A short PR interval, a delta wave, and attacks of paroxysmal tachycardia are the principal features of the WPW syndrome. A grossly irregular rhythm with bizzare QRS complexes at rates exceeding 180 per minute is one of the paroxysmal tachycardias characteristic of the WPW syndrome. This arrhythmia deserves special attention, because it is often misinterpretated as ventricular tachycardia. The interpretation is atrial fibrillation with varying degrees of ventricular fusion and phasic ventricular aberration. Prompt electrical cardioversion is indicated because, at times, ventricular fibrillation may result. Quinidine sulfate used prophylactically because of its negative dromotropic effect on the accessory pathway promotes A-V transmission via the A-V node.

Diagnosis, Differential↗
PubMedDate not supplied

Supraventricular tachycardia that mimics ventricular tachycardia. Part II.

Concealed WPW is a newly described clinical entity. The existence of this syndrome can be ascertained only through specific electrophysiologic intracardiac studies. From the clinical standpoint, the existence of an accessory unidirectional A-V pathway anatomically located in the general area of the Kent bundle should be suspected in those patients with chronic BBB who have recurrent paroxysmal atrial tachycardias initiated by sinus arrhythmias or premature atrial beats. It should be noted that functional BBB can occur following a premature atrial beat. This will also set the stage for the initiation of a reciprocating tachycardia in concealed WPW. Pharmacological therapy aimed at preventing the recurrent tachycardias is disappointing. Pacemaker implantation appears to be successful by rendering one of the bundle branches refractory through concealed conduction. With chronic block of the remaining bundle branch, the natural pathway of A-V conduction is totally blocked at a critical time during the tachycardias, thus eliminating the reciprocating tachycardia.

Adult↗
PubMedDate not supplied

A complication commonly overlooked.

Acute pulmonary embolism is one of the most underdiagnosed of the serious acute diseases that face the clinician. It is estimated to be the third most common cause of death in the United States. There is an increasing incidence of pulmonary embolism because of the growing size of the older population, greater number of extensive surgical procedures, increasing use of oral contraceptives, and prolongation of life in patients with metastatic malignancy. Pulmonary embolism is considered to be one of the most common single causes of death in the acute care general hospital. It is therefore important to appreciate that pulmonary embolism is a preventable and treatable disease.

Female↗