Use of Tinel's sign in carpal tunnel syndrome.
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Adult↗
Biomedical subjects
S W Asher
Publications and source records attributed to S W Asher.
Progressive hemifacial atrophy: report of three cases, including one observed over 43 years, and computed tomographic findings.
Three cases of progressive hemifacial atrophy were studied. In two patients, abnormalities were noted on the computed tomographic (CT) brain scan, whereas a third patient, with widespread cutaneous and neurologic involvement, had a normal CT scan. Patient 1, first described 44 years ago, had a neurologic disorder with features that have previously been associated with progressive hemifacial atrophy, but that have appeared years after the hemifacial atrophy ceased progression.
Adolescent↗
Tetrabenazine and movement disorders.
Based on blind review of videotape recordings, we analyzed the ability of tetrabenazine to suppress abnormal movements. The recordings were done before treatment, after 3 weeks on a stable dose (up to 200 mg daily) of tetrabenazine, and then after 3 weeks on placebo. Among the patients who completed the trial, improvement occurred in 6 of 8 with Huntington disease, 6 of 10 with tardive dyskinesia, and 3 of 8 with dystonic disorders. The drug was usually tolerated well.
Adult↗
Delayed sciatic palsy after total hip replacement: case report.
A 33-year-old woman with a 22-year history of juvenile rheumatoid arthritis developed right lower extremity sciatica 32 months after total hip arthroplasty. Physical examination and electromyography localized the abnormality to the sciatic nerve proximal to the midthigh level, involving the branch to the short head of the biceps femoris muscle. At surgical exploration, a sharp spur of methyl methacrylate, used to cement the acetabular prosthesis to the acetabulum, was found to have eroded through the lateral half of the sciatic nerve. The sciatica was relieved by neurolysis. The etiology of sciatic neuropathy after total hip replacement is reviewed.
Adult↗
Multiple cranial neuropathies, trigeminal neuralgia, and vascular headaches in sickle cell disease, a possible common mechanism.
Patients with sickle cell disease are subject to a variety of neurologic complications. A patient with sickle cell disease and rarely noted features or complications is described. The association of abnormal (sickle-type) hemoglobin (Hgb S) with trigeminal and facial neuropathies and vascular headaches appeared to coincide with reactivation of the hematologic disease.
Adult↗