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S Wansack

Publications and source records attributed to S Wansack.

3 recordsLinked to original sources

Hazard from an intense midrange impulse.

It has been hypothesized that the ear would become increasingly susceptible to impulses (gunfire) as the spectral peak of the impulse approached the frequency region where the ear was tuned best (about 4 kHz for the cat ear) [G. R. Price, J. Acoust. Soc. Am. Suppl. 1 62, S95 (1977)]. This prediction was counter to the predictions of the world's damage-risk criteria for impulse noise. It has been supported by experiments using exposures to 100-Hz and 800- to 1000-Hz impulses; but no test had been run at the point of predicted maximum susceptibility. In the present experiment, three groups of cats were exposed to 50 impulses produced by a primer explosion (spectral peak at 4 kHz) at peak levels of 135, 140, or 145 dB. Auditory thresholds were electrophysiologically measured from the vertex to 2-, 4-, 8-, and 16-kHz tone pips and losses were determined 30 min after exposure and more than 2 months post-exposure. Losses were greatest at 4 kHz, began to develop at 134-dB peak pressure, and the immediate losses grew at a rate of about 7 dB for every dB increase in peak pressure. About half of the loss measured immediately became permanent. The energy required to begin producing a permanent threshold shift was only about 0.07 J/m2, far lower than that required with continuous noises at lower sound pressures. The data were interpreted as supporting the original hypothesis of greater susceptibility in the midrange.

Acoustic Stimulation↗

The acoustic startle response and disruption of aiming: I. Effect of stimulus repetition, intensity, and intensity changes.

Three experiments examined the disruption of perceptual motor performance by intense noise bursts. Subjects aimed a rifle at a fixed target for 15-s periods separated by 15 s of rest. This cycle was repeated 30 times in each of two series separated by a 15-min rest, each series containing five noise bursts. The noise bursts disrupted aiming for 1-2 s, an effect that increased with sound pressure level for 110, 120, and 130 dB stimuli. There was no difference between stimuli with energy centered on 250 Hz as opposed to 800 Hz. The effect diminished over the five bursts within the first series (but not to zero) and did not recover in the 15-min rest period. Some subjects received three days of testing; in these cases the effect of the noise bursts partially recovered after rest intervals of 24 hrs and then seven days. Other subjects received 15 trials with 110-dB stimuli, then five more trials with 130-dB stimuli. The disruption of aiming by 130 dB stimuli was not reduced by prior exposure to 110-dB stimuli.

Adult↗

The acoustic startle response and disruption of aiming: II. Modulation by forewarning and preliminary stimuli.

Four experiments examined the disruption of rifle aim by intense noise bursts. In Experiment 1 a trigger pull was followed occasionally by a noise burst. Aiming was disrupted for 1-2 s, an effect that habituated within days and recovered between days. Expected stimuli were less disruptive than were unexpected stimuli. Experiment 2 demonstrated that weak auditory prestimuli 100 ms before unexpected intense sounds also reduced noise-produced errors. Experiment 3 showed that the intratympanic reflex had not played a protective role in this effect. Experiment 4 showed that a weak tactile prestimulus increased both a muscular measure of the acoustic startle reaction and the perturbing effect of the noise burst on motor performance. In general, conditions that affect the amplitude of the acoustic startle reflex similarly influence the disruptive effect of a noise burst on motor performance, but the two measures are not correlated in the detail necessary to suggest a causative relationship.

Adult↗