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T Beritić

Publications and source records attributed to T Beritić.

At least 19 recordsLinked to original sources
PubMed1993

[Analysis of patulin in apple juice].

A quantitative method for the determination of patulin in apple juice was examined. The procedure involved patulin extraction from apple juice with ethyl acetate, clean-up with column chromatography and preparative thin-layer chromatography. Fluorescent derivatives, obtained by exposure of patulin on chromatographic plates to concentrated ammonia fumes, permitted a convenient quantitative fluorodensitometric assay of patulin by means of the fluorescence quenching method. The detection limits were 200 ng of pure patulin and 100 micrograms of patulin per litre of apple juice. The recoveries of added patulin ranged from 78 to 110.4 percent, with a mean recovery of 97.8 percent.

Beverages↗
PubMed1989

Spinal origin of human lead neuropathy: this paper marks the 150th anniversary of Paralysie de Plomb ou Saturnine by L. Tanquerel des Planches.

One hundred fifty years ago a young but distinguished French scientist, L. Tanquerel des Planches, published a most comprehensive work dealing with almost every known clinical, epidemiological, and occupational aspect of lead poisoning, Traité des Maladies de Plomb ou Saturnines exposing in its second volume, Paralysie de Plomb ou Saturnine his invaluable experience on lead palsy. Among his ideas, based on a rather unique accumulation of human cases of lead palsy, the theory of the neurotoxic action of lead on the spinal cord is of outstanding importance. This site and the mode of action of lead have long been unsettled, although both the nineteenth century classics in neurology and the classics in industrial medicine of the first half of the twentieth century have contributed to solving the pathophysiology of lead palsy. The contributions from these reports, which concur with Tanquerel's assumptions, are presented and discussed. It must be emphasized, however, that only with the advent of electrophysiology has it become possible definitely to recognize the spinal cord involvement as the cause of lead neuropathy. The hypothesis of the axonal degeneration of the "dying back" variant, starting in the biochemical lesion of perikaria of the anterior horn cells in the spinal cord, is in full agreement with both the electromyographic signs of denervation and the electroneurographic normal range of conduction velocity. This presumptive conclusion confirming the spinal origin of human lead neuropathy is very much in line with the concepts of Tanquerel des Planches.

France↗
PubMed1989

[Mycotoxins and "yellow rain"].

A review is given of mycotoxins with a brief history of the role of these natural poisons in human pathology. Contributions of Croatian scientists to the field of research in mycotocixology are recorded. Opposite views and arguments are presented considering the technical possibilities and ethical responsibilities for abuse of mycotoxins as a war weapon especially with regard to their alleged use in the recent chemical warfare in the Middle and Far East. "Yellow Rain" is described as a natural phenomenon.

Chemical Warfare↗
PubMed1984

Lead neuropathy.

The still unexplained nature of the neurotropic action of lead has prompted this chronological survey of the course of development of the medical attitude towards the problems of lead neuropathy all along the centuries--from ancient times up to the present. Once a conspicuous, severe, and even frequent clinical type of plumbism, peripheral lead neuropathy has received due attention in the early classics (Tanquerel des Plances, Duchenne, Aran, Remak, Romberg, Erb, etc.) and of the pioneers in industrial medicine (Legge, Aub, Teleky, Hamilton). Even the modern era, however, has not come further than to state that lead produces different neurological effects in different animal species and even in humans--different patterns of neuromuscular involvement. With the advent of electrophysiology, conflicting and inconsistent findings have also appeared, particularly in regard to overt and latent lead neuropathies. Theories regarding the mode and site of the neurotoxic action of lead are reviewed and data which might be used as arguments both in favor and against are presented.

Aminolevulinic Acid↗