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T C Rotkis

Publications and source records attributed to T C Rotkis.

8 recordsLinked to original sources
PubMed1991

Pathologic bronchial vasculature in a case of massive hemoptysis due to chronic bronchitis.

The cause of bleeding in a patient with recurrent massive hemoptysis was not apparent after bronchoscopy and gross examination of the lobectomy specimen. Histologic submission of all major bronchi uncovered dilated, tortuous bronchial arteries just below the bronchial mucosa with sites of both current and healing arterial rupture. This bronchial arterial abnormality is common to several chronic pulmonary diseases, but is rarely diagnosed as a cause of massive hemoptysis. Careful pathologic examination of major bronchi in the setting of hemoptysis of unknown causation is recommended.

Aged↗
PubMed1984

Impaired gonadotropin responses to gonadotropin-releasing hormone stimulation in endurance-trained women.

The effects of endurance running on body composition, menstrual cycles, and gonadotropins were studied in 19 healthy, young, regularly menstruating women. Midfollicular plasma concentrations of unstimulated and gonadotropin-releasing hormone (GnRH)-stimulated luteinizing hormone and follicle-stimulating hormone were examined at baseline and after each subject's weekly mileage had increased 30 miles (delta 30) and 50 miles (delta 50) above baseline. Mean +/- standard error of the mean unstimulated luteinizing hormone and follicle-stimulating hormone did not change significantly. GnRH-stimulated luteinizing hormone was 76.3 +/- 22.0 micrograms/min/ml at baseline and declined to 20.2 +/- 4.5 micrograms/min/ml at delta 50 (P less than 0.02). GnRH-stimulated follicle-stimulating hormone was 28.4 +/- 7.0 micrograms/min/ml at baseline and declined to 9.6 +/- 2.1 micrograms/min/ml at delta 50 (P less than 0.02). There were no significant correlations between changes in body composition and changes in gonadotropin responses. Eighteen subjects developed oligomenorrhea.

Adult↗
PubMed1984

Thyroidal changes associated with endurance training in women.

The associations between endurance training, body composition, and the pituitary-thyroid axis were studied in 17 healthy, young women. Body composition and plasma concentrations of T4, T3, rT3, resin T3 uptake, TSH, and TRH-stimulated TSH were examined at baseline and after each subject's weekly distance had increased 48 km (delta 48) and 80 km (delta 80) above baseline. Total body weight did not change at delta 48 or delta 80. Mean (+/- SE) lean weight in kg increased from 42.9 +/- 1.2 at baseline to 44.8 +/- 1.2 at delta 80 (P = 0.002). We have reported previously that at delta 48 the subjects had evidence of mild thyroidal impairment, which consisted of decreased T3 and rT3, and an exaggerated TSH response to TRH. With more prolonged training (delta 48 to delta 80) there were significant increases in T4, rT3, and unstimulated TSH, while the ratios of T4/rT3 and T3/rT3 and the TSH response to TRH decreased significantly. Some of the thyroidal changes that occurred between delta 48 and delta 80 are similar to those seen in other stressful non-thyroidal conditions.

Adult↗
PubMed1983

Maximal exercise and residual lung volume: considerations for body composition analysis.

Pulmonary function was evaluated in 11 female cross country runners before and after maximal treadmill exercise. Hydrostatic weighing was also performed post exercise. Significant increases were found in both residual lung volume (X delta + 210 ml, 20.6%) and total lung capacity (X delta + 170 ml, 3.4%). These changes were not correlated to the intensity of the exercise. The magnitude of the change in residual lung volume resulted in a mean error of 12.1% in body composition prediction. Standardized testing orders are recommended.

Adult↗
PubMed1981

Specificity of power improvements through slow and fast isokinetic training.

College age males performed maximal two-legged isokinetic knee extensions three times per week for 6 wk at either 60 degrees/s (slow) or 300 degrees/s (fast) or both 60 and 300 degrees/s (mixed). The velocity specific and action specific (two-leg vs. one leg) improvements in peak torque (PT) were compared to a placebo group receiving low-level muscle stimulation. The slow group improved PT significantly (P less than 0.05) more than the placebo group only at its training velocity (60 degrees/s) and more so when the specific two-legged training action was mimicked (+32% with two legs vs. +19% with one leg). The mixed group enhanced PT by 24 and 16% at their respective training velocities of 60 and 300 degrees/s. These improvements were significantly larger than placebo and also significantly larger than the 9% improvement observed at the midvelocity of 180 degrees/s. The training specificity demonstrated by the slow and mixed groups suggest that neural mechanisms contributed to their improvements in power. This is supported by their unchanging muscle morphology. Training solely at 300 degrees/s (fast) however improved PT significantly more than placebo not only at the training velocity (+18%), but also at a slower velocity of 180 degrees/s (+17%). The fast group demonstrated a significant enlargement (+11%) of type II muscle fibers. These data suggest type II fiber hypertrophy to be a plausible mechanism for the nonspecific improvement of the fast group; however, a neurological adaptation that enhances power at and below the training velocity cannot be excluded.

Adult↗
PubMed1981

The effect of an acute bout of exercise on selected pulmonary function measurements.

The purpose of this study was to determine and follow the effect of an acute bout of exercise on residual volume (RV) and other pulmonary function measurements over a 24-h post-exercise period, and to evaluate the effect on subsequent body composition calculations. Selected pulmonary function measurements of 12 male college students were collected prior to exercise at 5, 15, and 30 min, and at 1, 2, and 24 h post-exercise. RV was significantly (P less than 0.01) increased 20.8% (260 ml), 16.8% (210 ml) and 12.0% (150 ml) at 5, 15, and 30 min post-exercise, respectively. Total lung capacity (TLC) was significantly (P less than 0.01) increased 2.7% (190 ml) and 2.3% (160 ml) at 5 and 15 min post-exercise, respectively. Subsequent body composition calculations, using the post-exercise RV data, resulted in significantly (P less than 0.01) lower relative body fat measures, as compared to the pre-exercise value, through 30 min of recovery. In conclusion, our data demonstrate apparent increases in RV and TLC immediately following maximal treadmill exercise; these measurements remained significantly increased over pre-exercise values, through 30 and 15 min of recovery, respectively. It is recommended that the RV measurement and the actual hydrostatic weighing procedure not be separated by an acute bout of exercise, and that protocols for long-term studies be standardized.

Adult↗