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Biomedical subjects

T O Rørvik

Publications and source records attributed to T O Rørvik.

8 recordsLinked to original sources
PubMed1983

Severe iron deficiency causing loss of ring sideroblasts.

A patient with primary sideroblastic anaemia was observed for more than 10 years with 50% ring sideroblasts in the bone marrow and a Hb of 84-109 g/l. Iron deficiency then developed with loss of stainable iron in marrow particles (reticuloendothelial iron) and loss of all ring sideroblasts. When iron tablets had brought the iron stores in the bone marrow up to subnormal levels, the Hb was over 110 g/l, somewhat higher than it had been when the iron stores were increased. The ring sideroblasts were again raised to more than 50%.

Anemia, Sideroblastic↗
PubMed1981

Differing form of variant form of hypergranular promyelocytic leukaemia (M3), or transition between M3 and monocytic leukaemia?

A 17-year-old woman had acute myeloid leukaemia (AML) with a leucocyte count of about 100 x 10(9)/1. The great majority of the leucocytes were immature. About half of them were without granules; in the other half the granules were usually scanty, but occasionally numerous and rather coarse. A few cells had Auer rods arranged in bundles. The patient had uncompensated disseminated intravascular coagulation (DIC) with a severe tendency to bleed. Everything fitted with variant hypergranular promyelocytic leukaemia, except for the staining of 30% of the immature cells by Löffler's unspecific esterase with alpha-naphthyl acetate. The patient may, therefore, be classified as either a variant of variant form M3, or as a transition between M3 and acute monocytic leukaemia.

Adolescent↗
PubMed1980

Abnormal pattern of the rough endoplasmic reticulum of plasma cells in multiple myeloma with multiple concentric lamellar bodies and "single sac loops".

A patient with IgG myeloma showed a highly abnormal pattern of the rough endoplasmic reticulum (RER) with multiple concentric lamellar bodies and "single sac loops". This abnormal pattern might be due to either a complete rearrangement of the architecture within the cell, and/or a pathology in the wall of the sacs of RER.

Endoplasmic Reticulum↗