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T Rabinowicz

Publications and source records attributed to T Rabinowicz.

At least 37 records · Page 2Linked to original sources
PubMed1978

Postnatal development of the mouse cerebral neocortex. IV. Evolution of the total cortical volume, of the population of neurons and glial cells.

The total neocortical volume delimited between precise anatomical features was measured. Serial frontal sections cresyl-violet stained were measured with plainimetric methods and the total volume evaluated at 5, 10, 30, 60 and 180 days. The total volume was used to obtain the total number of neurons and glial cells at each age using our quantitative data (published in our previous papers). The evolution of the cellular densities were studied in the sensory and motor areas 10--4--3 and 2 (Leuba and coll. 1977) and in the visual and auditory areas 17--18a--18--41 and 20 (Heumann anc coll. 1977) in the mouse cerebral neocortex. 1. The total neocortical volume increases rapidly between 5 and 10 days, less between 10 and 30 days, slightly decreases at 60 days and reincreases at 180 days. 2. Between 5 and 30 days, the loss of cortical neurons is of about 30% by neuronal death. 3. The total number of glial cells doubles between 5 and 180 days. The increase is progressive through the development.

Animals↗
PubMed1977

Cerebral coenurosis. Report of a case with arteritis.

A case of the unusual clinical condition of cerebral coenurosis, the unique parasitic disease that is localized only in the CNS, is reported. The patient had repeated attacks of transient hemiparesis due to intracranial arteritis which was demonstrated by several angiographic studies. The CSF showed a discreet lymphocytosis and increased immunoglobulins. This inflammatory reaction which preceeded the manifestations of the intracranial mass can be explained by the parasitic foreign bodies, the liberation of somatic and metabolic parasitic toxins and the immunological reaction of the organism. The arteritis of the basal intracranial vessels was probably secondary to the inflammatory reaction of the leptomeninges. In presence of the rare association of meningovascular disease and an expanding intracranial lesion a parasitic condition due to a larval stage of Cestodes (Hydatidosis, Cysticercosis, Coenurosis) should be always considered.

Adult↗
PubMed1977

Intrauterine brain death. Neuraxial reticular core necrosis.

Report is given on the first example in which the clinical and electrographic criteria of brain death were obtained at birth. Global destruction of the central nervous system of a type seen only with anoxia and circulatory failure had occurred in utero without appreciable disturbance of maternal health. Neuropathologic reaction in fetal tissues was identical with that which has been observed in the child or adult in the brain death syndrome. Functional disturbance of cerebral spinal circulation of sufficient degree to destroy neuronal tissue had happened without change in maternal circulation and without demonstrable lesions in the placenta or the cardiovascular apparatus of the fetus.

Brain Death↗
PubMed1977

Postnatal development of the mouse cerebral neocortex. I. Quantitative cytoarchitectonics of some motor and sensory areas.

Quantitative data have been obtained (numbers of neurons and glial cells in a cube of 0.000 25 mm3, thicknesses of cortical layers, nuclear diameters of neurons) on Cresyl Violet stained sections from the six cortical layers of the Swiss Albinos mouse brain through the postnatal development at 5, 10, 30, 60 and 180 days. 1) Normative data of the cortical cell population are now available from motor areas 4 and 10 and sensory areas 3 and 2 according to KRIEG (1946). The evolution of the neuronal densities through time was obtained and was taken as a criterion of maturation for the different layers in the studied cortical areas. 2) Areas 4, 10, 3 and 2 have been described histologically at all the mentioned ages. 3) The nuclear diameter of the neurons increases in all the layers between 5 and 10 days and less between 10 and 30 days. It remains stable from 30 days on. 4) At adult age the sensory areas 3 and 2 show a greater thickness of the group of supragranular layers II, III and IV than of the infragranular layers V and VI together. Layer V shows the same thickness in areas 2, 3 and 4. The same is true for layer VI in the same areas. In area 10 layers V and VI are thicker than in areas 2, 3 and 4. At younger ages (5 and 10 days) the relative thicknesses of the different layers are of the same order but each layer is thinner. 5) The neuronal density shows the same type of development in the four studied areas. Maturation in terms of decrease of neuronal densities is very rapid between 5 and 10 days and slows down till 30 days with speeds depending on the layers. Layers I, V and VI show faster and earlier maturation than layers II, III and IV. 6) The glial density is mostly stable through the development. This does not mean that there is no glial proliferation. 7) The delineation between the different areas according to KRIEG's (1946) atals and/or CAVINESS's (1975) publication are discussed taking into account our quantitative data.

Animals↗
PubMed1976

[Amnesia syndrome due to tumor infiltration].

The authors present their anatomo-clinical observation of amnesic syndrome occurring with brain tumoral infiltration. On the clinical level, the disorder is characterized by a memory encoding deficit. Anatomical examination shows a bilateral lesion of the mamillary bodies.

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