The response of single human cells to zero gravity.
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Amino Acids↗
Biomedical subjects
T Rogers
Publications and source records attributed to T Rogers.
Clinical results of bone marrow transplantation in aplasia. Report of Westminster Hospital bone marrow transplant team [proceedings].
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Anemia, Aplastic↗
Gentamicin resistance in Staphylococcus aureus.
A clinical isolate of Staphylococcus aureus was resistant to gentamicin, kanamycin, and tobramycin by virtue of the production of a drug-inactivating enzyme. Attempts to establish the cellular location of the genetic determinants for enzyme synthesis have given equivocal results.
Anti-Bacterial Agents↗
Experimental Candida albicans infection in conventional mice and germfree rats.
Swiss-Webster white mice were intravenously infected with various doses of Candida albicans, and the viable units in their spleens, livers, lungs, and kidneys were determined at various intervals after challenge. The results showed that C. albicans multiplied to a greater extent in the kidneys of mice than in their spleens, lungs, or livers. The infection in mice was chronic; increasing numbers of C. albicans were observed in their kidneys until about 17 to 24 days postchallenge. Clearance of C. albicans from infected kidneys was not symmetrical, since the number of viable C. albicans in one kidney did not coincide with the viable counts observed in the opposite kidney of that same animal. Male and female mice did not differ in their overall susceptibility (50% lethal dose test) or in the number of viable C. albicans in the kidneys at various time intervals after infection. C. albicans also multiplied in the kidneys of germfree rats; however, the peak of the C. albicans infection in their kidneys occurred earlier than in those of conventional mice.
Animals↗
Effect of potassium-sparing diuretics on the renin-angiotensin-aldosterone system and potassium retention in heart failure.
The renin-angiotensin-aldosterone system and electrolyte levels in 11 patients with heart failure controlled on digoxin and frusemide were investigated after separate periods of Slow K, spironolactone, and amiloride therapy. When spironolactone or amiloride replaced Slow K, distinct parallel increments in the levels of renin, angiotensin II, and aldosterone resulted. Though plasma potassium was generally higher after spironolactone and amiloride than after Slow K, exchangeable potassium was similar with the three regimens. There was no significant relation between plasma potassium and concurrent exchangeable potassium.
Adult↗