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Biomedical subjects

T Sanjo

Publications and source records attributed to T Sanjo.

At least 19 recordsLinked to original sources
PubMed1998

Maxillofacial morphology and masseter muscle thickness in adults.

The aim of this study was to investigate how the thickness of the masseter muscle relates to the maxillofacial morphology, including the thickness of alveolar process in the mandibular incisor region, and the thickness of the mandibular symphysis. The subjects consisted of 80 adult male volunteers (mean age: 23 years 8 months). The relationship between masseter muscle thickness and the maxillofacial skeleton was investigated by measuring the former by ultrasonography and the latter by roentgenographic cephalometry. The data were initially analysed using a multiple regression analysis. Thereafter, correlation coefficients were obtained by a simple regression analysis. The following results were found: 1. The thickness of the masseter muscle (mean +/- SD) was 15.8 +/- 3.0 mm in the relaxed state and 16.7 +/- 2.7 mm at maximal clenching. 2. Masseter muscle thickness was negatively correlated with the mandibular plane angle. 3. Masseter muscle thickness was positively correlated with the mandibular ramus height (Cd-Go), and the thickness of the alveolar process and that of the mandibular symphysis. It is therefore suggested that masticatory function influences the morphology of the mandible.

Adult↗
PubMed1986

Hemodiafiltration treatment of deep hepatic coma by protein passing membrane: case report.

Based on the assumption that middle molecules that induce hepatic coma occur in the plasma of patients with fulminant hepatic failure (FHF), the authors have developed a hemodiafiltration (HDF) method using a large-pore poly(methylmethacrylate) (PMMA) membrane that allows proteins to pass through. PMMA HDF resulted in complete recovery of deep coma and long-term survival in a patient with severe FHF. Although a large-scale controlled trial is needed, PMMA HDF is believed to be promising as a future artificial liver support.

Adult↗
PubMed1980

Effect of spontaneous portal-systemic shunting on plasma insulin and amino acid concentrations.

To elucidate the relative contribution of parenchymal liver damage and spontaneous portal-systemic shunting to the reduction of peripheral insulin degradation rate and the decrease in plasma concentrations of three branched chain amino acids (valine, leucine, and isoleucine), plasma insulin, C-peptide, and amino acid concentrations were measured during oral glucose tolerance tests in 17 patients with liver cirrhosis, 10 with idiopathic portal hypertension, 5 hospitalized controls, and normal subjects. None of the patients had evidence of hepatic encephalopathy. Patients with idiopathic portal hypertension had histologically minimum hepatic fibrosis in spite of the existence of extensive exophageal varices. The molar ratio between plasma concentrations of C-peptide and insulin was significantly decreased in patients with cirrhosis, but not in those with idiopathic portal hypertension. In both patients with cirrhosis and idiopathic portal hypertension, the three branched chain amino acid levels were significantly decreased and the molar ratio between the concentrations of the three branched chain amino acids and two aromatic amino acids (tyrosine and phenylalanine) were markedly reduced. These results suggest that spontaneous portal-systemic shunting does not primarily contribute to the reduced degradation of insulin, but has a close relationship with the decrease in branched chain amino acid levels and in the molar ratio of plasma amino acids. In addition, the present data indicate that decreased branched chain amino acid levels in patients with cirrhosis is not merely ascribed to hyperinsulinemia and that the decrease in the molar ratio of plasma amino acids is not specific to the presence of hepatic encephalopathy.

Adult↗