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Biomedical subjects

T Wensing

Publications and source records attributed to T Wensing.

137 records · Page 8Linked to original sources
PubMed1980

[Some clinical biochemical features of 'tying up' in horses (author's transl)].

The changes in the activities of lactate dehydrogenase (LDH) and creatine phosphokinase (CPK) in de blood of thirty-three horses with 'tying up' were compared. The extent to which the serum enzymes LDH, CPK and glutamic oxaloacetic transaminase (GOT) and the changes in the activities of these enzymes after suitable labour can be used in the diagnosis of 'tying up' and in following the recovery of patients was studied.

Animals↗
PubMed1978

[Incidence of Clostridium botulinum in the rumen contents and faeces of cattle fed brewers' grains naturally contaminated with Clostridium botulinum (author's transl)].

The number of Clostridium botulinum type B organisms excreted by cattle fed brewers' grains in which these organisms were found to be present and the period for which they were excreted, were studied. Large numbers (10(5) - 10(7) per gramme) of these organisms were detected in the rumen contents and faeces of the animals. When feeding brewers' grains was discontinued, Cl. botulinum type B was still detectable in the faeces for a considerable period (greater than eight weeks). There was evidence to suggest that the number of Cl. botulinum organisms multiplies in the gastrointestinal tract of cattle.

Animal Feed↗
PubMed1978

[Food poisoning in cattle caused by ingestion of brewers' grains contaminated with Clostridium botulinum type B (author's transl)].

In 1977, outbreaks of disease associated with serious losses occurred on twenty cattle farms. This disease was due to the fact that the animals had been fed brewers' grains. The clinical picture was marked by anorexia, profuse salivation, regurgitation and dehydration. The course of the disease varied with the severity of the attack. Toxicological tests were negative. Cl. botulinum type B as well as toxin type B were found to be present in the brewers' grains fairly often. As a rule, Cl. botulinum was also detected in the rumen contents and faeces. The treatment of the animals is described; this resulted in recovery in a number of cases. When experimental animals were fed brewers' grains from farms on which outbreaks had occurred, this induced similar clinical pictures. The results of studies on the aetiology and pathogenesis are discussed. It is concluded that the clinical picture was an atypical form of botulism caused by Cl. botulinum type B. Possible causes of this abnormal clinical picture are discussed in greater detail.

Animal Feed↗
PubMed1975

[Estimation of the concentration of urea in the blood of horses, cattle, goats and dogs using the "Merckognost Harnstoff" method compared with an enzymatic, photometric method (author's transl)].

To examine the suitability and reliability in field use of the "Merckognost Harnstoff" method in estimating the concentration of urea in the blood of horses, cattle, goats and dogs, the levels determined by this procedure were compared with those determined by an enzymatic (urease) photometric method widely used in laboratories. It was concluded from the results obtained that estimation using the "Merckognost Harnstoff" is sufficiently reliable for the rapid assay of urea in the blood under field conditions.

Animals↗
PubMed1975

[Occurrence, diagnosis and course of chronic hepatitis in weaned lambs of a flock of Texel sheep (author's transl)].

A disease of weaned lambs is described, which is attributable to chronic hepatitis marked by impaired secretion of cholephils in the acute or early stage and by inadequate growth due to persistent dysfunction of the liver during the subsequent or chronic stage. Regardless of its duration, the disease may be diagnosed in the living animal by determining the SDH and gammaGT activity levels, the total serum protein, the serum iron concentration and the serum iron-binding capacity.

Alcohol Oxidoreductases↗
PubMed1975

Hyperlipoproteinaemia in ponies: mechanisms and response to therapy.

(I) The lipid and lipoprotein concentrations in sera of 4 healthy and 9 hyperlipaemic patients were determined. From the results of the analyses, it is suggested that three types of hyperlipoproteinaemia can be distinguished in ponies: (a) Type 1, characterised by a very pronounced increase in only the very low density lipoproteins (VLDL); (b) Type 2, in which there is a pronounced increase in the chylomicrons (Chylo) and the very low density lipoproteins (VLDL); (c) Type 3, which can be the result of fasting and is characterised by a moderate increase in the concentration of chylomicrons and a greater increase in very low density lipoproteins. It is suggested that hyperlipoproteinaemia in ponies can be classified on the basis of electrophoretic patterns. (II) To gather more information on the metabolism of extra offered lipids, blood from ponies suffering from two different types of hyperlipoproteinaemia was transfused into normal ponies and the changes in lipid and lipoprotein concentrations in the blood serum were followed. The reactions were different. When heparin was used as anticoagulant a great increase in chylomicrons and a significant increase in very low density lipoproteins in a healthy pony was observed. When sodium citrate was used as anticoagulant a clear increase in the VLDL fraction only was noticed.

Animals↗
PubMedDate not supplied

The effect of undernutrition on haematological and serum biochemical variables in wild boar (Sus scrofa).

The effect of induced long term undernutrition on the blood composition of wild boar was studied. During the first weeks, the activity of plasma alkaline phosphatase decreased, while creatinine concentrations increased, and the ratio urea/creatinine decreased, suggesting a renal mechanism as well as recycling of nitrogenous waste products to conserve nitrogen. During the last weeks, haemoglobin and packed cell volume decreased, suggesting protein or mineral deficiencies, and the ratio urea/creatinine increased, indicating substantial endogenous protein catabolism.

Alkaline Phosphatase↗