Biomedical subjects

Toyotaka Aiba

Publications and source records attributed to Toyotaka Aiba.

4 recordsLinked to original sources

PubMed2004

Bilateral medial medullary infarction due to bilateral vertebral artery dissection.

We describe a 52-year-old woman who experienced transient motor weakness and numbness of the left extremities and presented 2 days later with severe hemiparesis and sensory impairment of the right extremities and right lingual palsy. Magnetic resonance imaging (MRI) revealed bilateral upper medial medullary infarction, primarily in the left ventral portion. The findings of both three-dimensional (3D) computed tomographic and conventional angiography suggested dissection of both intracranial vertebral arteries (VAs). Medial medullary infarction is generally caused by atherosclerosis within a VA or anterior spinal artery. This is the first report of bilateral medial medullary infarction due to dissection of both intracranial VAs.

Brain Stem Infarctions↗

PubMed2003

Unilateral oculomotor nerve paresis associated with anterior communicating artery aneurysm rupture--two case reports.

Two cases of complete unilateral oculomotor nerve palsy occurred after subarachnoid hemorrhage (SAH) due to a ruptured anterior communicating artery aneurysm. A 61-year-old female suffered left oculomotor nerve paresis after mild SAH. This paresis was probably related to pre-existing oculomotor nerve stretching caused by abnormal positioning of the posterior cerebral and superior cerebellar arteries in the premesencephalic cistern. A 70-year-old female suffered right oculomotor nerve paresis after severe SAH. Elevated intracranial pressure might have caused this paresis, but the reason for the unilateral occurrence was undetermined. Both patients were treated by clipping of the aneurysm, and the signs of oculomotor nerve paresis gradually resolved. A pattern of pupil-sparing paresis was observed during the early recovery stage in both patients.

Aged↗

PubMed2003

[Ruptured frontopolar artery aneurysm: two cases].

We report two very rare cases of subarachnoid hemorrhage due to a ruptured frontopolar artery aneurysm in the cingulate sulcus. Only two surgically treated cases have been reported. Case 1: A 69-year-old woman suffered headache and was admitted to our hospital the next day. Computed tomography (CT) revealed focal subarachnoid hemorrhage (SAH) in the interhemispheric fissure. Cerebral angiography on admission failed to show an aneurysm, but follow-up study on day 14 revealed a small aneurysm in the cingulate sulcus at the origin of the frontopolar artery arising from the callosomarginal trunk. The aneurysm was successfully clipped on day 15 and the post-operative course was uneventful. Case 2: A 43-year-old man was transferred to our hospital complaining of severe headache. CT revealed diffuse SAH in the basal and interhemispheric cistern. Cerebral angiography showed an aneurysm at the origin of the callosomarginal trunk arising from the anterior communicating artery complex and another at the callosomarginal-frontopolar bifurcation in the cingulate sulcus. Both aneurysms were successfully clipped on the day of admission, and the frontopolar artery aneurysm was shown to be the cause of the SAH.

Adult↗

PubMed2002

[Shotgun injury of head and neck: case report].

A 48-year-old male was hit by a shotgun blast from behind while he was hunting. He was only 7 meters away when one of his colleagues pulled the trigger accidentally. About 180 lead pellets penetrated his head and neck. When he was brought to our hospital, he was alert and complained of occipital and nape pain, but had no apparent neurological deficits. We administered 200 mg of dimercaprol immediately and removed 77 pellets by midline suboccipital incision on the next day. Afterwords, we performed four minor operations using a fluoroscope, and removed another 70 pellets by day 29. In addition, we administered a 100 mg x 3 dose of dimercaprol every other week. He was discharged 41 days after the accident without any acute signs of lead poisoning. Though serum lead level did not rise to critical level, EDTA was continued after his discharge to prevent delayed lead poisoning from the retained pellets. Since we seldom encounter this type of patient in Japan, delayed lead poisoning by shotgun injury was discussed with reference to the literature.

Chelating Agents↗