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Biomedical subjects

V Becker

Publications and source records attributed to V Becker.

149 records · Page 9Linked to original sources

Groove pancreatitis.

Groove pancreatitis is characterized by the formation of a scar plate between the head of the pancreas and the duodenum, and is by no means a rare entity. In our material comprising surgical specimens, it was found to be present in 19.5% of the cases. This form of pancreatitis is "special" only on account of the topography of the scar tissue. Scarring of the groove leads to complications that are also determined by the topography: disturbance of the motility of the duodenum, stenosis of the duodenum, and tubular stenosis of the common bile duct, which occasionally extends to obstructive jaundice. In the "pure" forms of groove pancreatitis the main pancreatic duct is not involved, and this fact has a certain significance in the differential diagnosis vis-à-vis carcinoma of the head of the pancreas, since signs of stenosis with no involvement of the rest of the pancreas are, in the first instance, suspicious for carcinoma and provide the indication for surgery. A knowledge of the existence of groove pancreatitis suggests a differential-diagnostic alternative. In terms of etiology, pathogenesis, and the pathological/anatomical picture, groove pancreatitis is "special" only on account of its topographic aspects.

Chronic Disease

[Morphologic placenta findings in clinico-serologically verified and suspected rubella infection in the 2d half of pregnancy].

Rubeola is the congenital infection with the highest rate of embryonal damage. The hematogenous transplacental way of infection also causes morphological changes at the vessels of the placental villi. We examined 38 placentas from up to the 44th week of gestation and the fetus and children belonging to them. We regarded the clinical and serological findings. 6 out of 16 placentas from up to the 18th week of gestation (37.5%) showed endangitis obliterans. Two cases showed changes of the lenses. (Group I). In 10 out of 12 cases from the 19th to the 36th week of gestation (Group II) we found morphological changes in the vessels of the placental villi. The children belonging to them showed nonspecific malformations. From the 37th week of gestation (Group III) half of the placentas was histologically conspicuous. The children did not show malformations specific for rubeola. We refer to the possibilities and limitations of performing light microscopic diagnosis in placenta when there is the suspicion of rubeola infection, intrauterine death of the fetus and postnatal delay of the child's development.

Antibodies, Viral