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Biomedical subjects

V M Elizarova

Publications and source records attributed to V M Elizarova.

9 recordsLinked to original sources
PubMed2006

[Helicobacter pylori-associated pathology of oral cavity in children (clinical-laboratory study)].

As the result of the study of stomatological status indices in children with gastroduodenal pathology associated with Helicobacter pylori it was established that caries incidence in children did not depend upon contamination while caries prevalence in examined children unlike caries incidence was associated with HP-status of the oral cavity. Patients with concomitant gastroduodenal pathology had frequently periodontal disease (PD). In children with chronic antral gastritis associated with Helicobacter pylori clinical manifestations were poor and tended towards inflammatory process chronicity. All the patients had chronic catarrhal gingivitis. In children with Helicobacter pylori associated pathology of GIT it proceeded with 100% contamination of gingival mucous membrane by Helicobacter pylori as shown by bacterioscopic study.

Adolescent↗
PubMed2002

[Calcium homeostasis disorders in children with multiple caries].

A saliva of the examined children of 3, 6, 12 years old with intensive caries are characterized by a changing of calcium homeostasis which is accompanied with redistribution of calcium fractions in a mixed nonstimulation saliva. In this case the concentration of Ca2+ is raisign considerably while the concentration of general calcium is raising slightly.

Calcium↗
PubMed1998

[Primary anodontia in X-linked hidrotic ectodermal dysplasia].

Nine patients with congenital deciduous and permanent adentia are described for the first time in Russia. Published reports are reviewed and recommendations on the DNA and prenatal diagnosis offered, which permit detecting women at a high risk of giving birth to handicapped children.

Adult↗
PubMed1997

[Ionized calcium in the saliva of children with multiple caries].

Ionized calcium and total content of all Ca fractions were measured in mixed saliva and blood serum of children aged 4 to 12 with solitary carious lesions or multiple caries. The level of ionized Ca was much higher in multiple caries than in individual lesions, and the content of bound Ca (protein-bound and in complex with other compounds) was decreased. The authors propose that recharging of salivary proteins caused by acidification of the saliva and dental deposit due to production of lactate and other organic acids is mainly responsible for redistribution of Ca fractions in mixed saliva. Ca release which results from demineralization of teeth, changed rate of salivary secretion and permeability of the blood-saliva barrier, and other factors may also contribute to redistribution of Ca fractions in the saliva, which plays an important role in the pathogenesis of caries.

Calcium↗