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V N Anokhin

Publications and source records attributed to V N Anokhin.

At least 19 recordsLinked to original sources
PubMed1996

[Formation and development of rheumatology in Russia].

The formation and development of rheumatology in Russia go back to February 4, 1928, the date of setting up the Committee for Rheumatism Study and Control in the USSR at the RSFSR People's Commissariat of Health. Later the Committee was renamed the All-Union Antirheumatic Committee. The paper outlines the contribution of those who initiated to establish the Committee (M. P. Konchalovsky, G. M. Danishevsky, and others), analyzes the organizational structures for developing rheumatology and the evolution of teaching of rheumatism and other rheumatic diseases. It also gives some names of Russian scientists who made a great contribution to the development of rheumatology (V. T. Talalayev, M. A. Skvortsov, A. A. Kisel, A. I. Nesterov, N. D. Strazhesko, M. M. Diterikhs, M. G. Astapenko, V. A. Nasonova, etc.) and makes a brief review of the present status of rheumatology.

History, 20th Century↗
PubMed1993

[Evaluation of the severity of aortic stenosis using 2-dimensional Doppler echocardiography].

Hemodynamic evaluation of aortic ostial stenosis (AOS) was made in 89 patients at Doppler echocardiography. Maximal circulation rate (MCR) through the aortic valve averaged 3.47 +/- 0.073 m/s, maximal transaortic pressure gradient (TPG) made up 49.97 +/- 2.11 mm Hg, the aortic ostium area (AOA) amounted to 0.85 +/- 0.031 sm2. It was established that AOA evaluation is most reasonable, as MCR and TPG vary with cardiac output. Especially desirable this measurement is believed in patients with TPG under 64 mm Hg and small left ventricular ejection. Mitral regurgitation is a frequent finding in AOS patients. Unless calcinosis of the mitral ring, mitral valve affection would be absent. In mitral regurgitation the disease took a more severe course, the patients having reduced AOA and left ventricular ejection, though larger end-diastolic diameter and end-diastolic volume. The emergence of mitral regurgitation in AOS is a result of left ventricular hypertrophy and dilatation suggesting a low compensatory reserve of the myocardium.

Aortic Valve↗
PubMed1991

[A comparative assessment of the efficacy of thiophosphamide electrophoresis and ultrasound in the combined therapy of reactive arthritis].

The results of the treatment of patients suffering from rheumatoid arthritis with ultrasound (group I), thiophosphamide electrophoresis (group II), ultrasound and thiophosphamide (group III) and with medicamentous agents alone (group IV) are compared. The positive therapeutic effect was recorded in patients of all 4 groups, with that effect being most remarkable in group III.

Adult↗
PubMed1990

[The assessment of left ventricular diastolic function in patients with aortic defects].

Thirty patients with aortic heart disease and 10 healthy persons were examined for diastolic function of the left ventricle using two-dimensional Doppler echocardiography. The decline of the rate and volume of early diastolic filling, the rise of the filling during the atrial systole were revealed in 60% of the patients with aortal disease. The decrease of the ejection fraction of the left ventricle was noted in 23.3% of the patients. All the patients with aortic disease were distributed into 2 groups depending on the presence (group II) or lack (group I) of mitral regurgitation. Addition of mitral regurgitation in patients with aortic disease masked the deranged filling of the left ventricle and interfered with the diagnosis of diastolic dysfunction. A reverse moderately pronounced relationship (r = -0.56) has been discovered between the myocardial mass and impairment of the diastolic filling.

Adult↗
PubMedDate not supplied

[Detection of B-cell marker of rheumatism using monoclonal antibodies D8/17 in the families of patients with rheumatism].

To study hereditary disposition for rheumatism (R) 141 persons including 26 probands of patients with R, 79 relatives and also 12 newborns of mothers with R were examined. The carriage of the B-cell markers was studied by means of antibodies D8/17. The B-cell marker was revealed in 96.2 per cent probands and in 41.4 per cent relatives. Among the female relatives with R the B-cell marker carriage was revealed in 51.2 per cent, among the male relatives in 25.9 per cent. A high carriage of the given marker in children and young people aged 4-34 born of mothers with R was noted (66.7 per cent). Among the newborn no carriers of the B-cell marker were detected. A conclusion has been made of the participation of the B-cell marker in the formation of hereditary predisposition for R.

Adult↗
PubMedDate not supplied

[Detection of B-cell marker of rheumatism using monoclonal antibodies D8/17 in families of patients with rheumatism (report 1)].

Results of clinico-genealogical and immunogenetic examination of 220 persons including 150 patients suffering from rheumatism and members of their families have been presented. The object of the examination was a B-cell marker carriership to be determined by means of monoclonic antibodies D8/17 with the use of the microlymphocytotoxic method. The marker carriership was detected in 96 per cent of probands-patients with rheumatism and in 40.3 per cent of their relatives and in 6.7 per cent of healthy individuals of the control group. A possible contribution of the B-cell marker to rheumatism liability was suggested. A further study of the role of the given marker in rheumatism has been found advisable.

Adult↗