Biomedical subjects
Publications and source records attributed to V Z Alesov.
An examination was performed in 15 patients who had developed pronounced osteomalacia following gastric resection. The discussion covers potential pathogenetic mechanisms responsible for bone tissue metabolism disorders and approaches to correction thereof.
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Electrolytic imbalance is a frequent finding in malabsorption syndrome. Derangement of calcium metabolism present clinically in different variants is most serious. Some patients develop hypocalcemia manifesting clinically with specific myasthenia, paresthesias, convulsions, hemorrhages, etc. In other variants severe skeletal lesions are seen which may become dominating in the clinical picture though hypocalcemia was absent. Investigation of calcium metabolism, hormonal profile (parathormone, in particular) in malabsorption syndrome can prognosticate and prevent the onset of osteomalacia.
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The method of extracorporal hemosorption was used by the authors first in the clinical practice for the preoperative preparation of patients with thyrotoxicosis. Oxygenated SKN carbons were used as sorbents. The hemosorption was used in 16 patients with thyrotoxic goiter by the method developed in the clinic. The authors make a conclusion that hemosorption is an expedient method for the preoperative preparation of patients with a toxic goiter.
Serum protein-bound iodine (PBI), total blood thyrotoxin (T4), triiodthyronine (T3), coefficient of thyrotoxin efficacy (CTE), serum thyrotoxin-binding capacity (TBC) or some of these indices were determined in 44 patients with nodular toxic goiter with thyrotoxemia. An increase in T3 level was revealed in 27 of 35 of the patients; T3-thyrotoxicosis was diagnosed in 18 of these in whom an increase of the T3 level was combined with normal T4 or normal CTE content and an unchanged TBC. The symptoms of T3-thyrotoxicosis displayed no significant differences from the symptoms of thyrotoxicosis caused by the excess of thyroxin. In many of the patients T3-thyrotoxicosis coursed for a long time with but a few symptoms. Loss of weight in this form of thyrotoxicosis was much less frequent than in "thyrotoxin'' thyrotoxicosis. PBI was elevated in 12 of 44 patients, and total T4 and CTE -- in 10 of 35. These methods do not permit to diagnose hyperthyroidism in many of the patients with nodular toxic goiter in which T3-thyrotoxicosis is of frequent occurrence.
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