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Biomedical subjects

W Babisch

Publications and source records attributed to W Babisch.

At least 19 recordsLinked to original sources
PubMed2004

[Environmental Survey for Children-- the environmental module of KiGGS. I. Design and research program].

The German Environmental Survey for Children (GerES IV) is the environment-oriented module of the National Health Interview and Examination Survey for Children and Adolescents (KiGGS) which is being performed nationwide in Germany. From 2003 to 2006, a random subsample of 1800 children aged 3-14 years is being studied with regard to their body burden and health impairments linked to housing conditions and the personal environment- and health-relevant behaviour. The basic study programme includes the analysis of blood, urine, tap water and house dust as well as the application of an extensive questionnaire. The data gained from this population sample, which is representative for Germany's children, are the basis for deriving reference values to characterise the background exposure of children aged 3-14 years. Trends over time can be detected and the success of environmental policies verified by comparing the data with those of the German Environmental Survey 1990/92 (GerES II), also conducted in close cooperation with the National Health Survey, which included children aged 6-14 years. By linking the data from the Environmental and the Health Surveys, health-relevant environmental exposures can be detected and different scientific hypotheses can be tested. The main subjects that are being dealt with using subcollectives of GerES IV are 'VOC and eye and nasopharynx irritation', 'indoor allergens and allergic diseases of the respiratory system', 'chromium, nickel, fragrances and contact allergens', and 'noise, hearing capacity and stress hormones'.

Adolescent↗
PubMed2003

Health status as a potential effect modifier of the relation between noise annoyance and incidence of ischaemic heart disease.

AIMS: Traffic noise is a psychosocial stressor. Epidemiological studies suggest chronic noise stress to be a risk factor for cardiovascular disorders. METHODS: In a prospective cohort study, the association between annoyance and disturbances due to road traffic noise and the incidence of ischaemic heart disease (IHD) was studied in 3950 middle aged men. RESULTS: Depending on the questionnaire item, non-significant odds ratios for IHD incidence ranging from 0.9 to 1.4 were found for the highly noise annoyed/disturbed subjects when compared with the less annoyed/disturbed subjects, over the six year follow up period. However, this relation was strongly modified by the prevalence of pre-existing chronic diseases. In subjects free of any chronic disease at the beginning of the follow up, significant odds ratios between 1.7 and 3.0 were seen. In the subgroup with chronic diseases no such noise effects were seen. This surprising result of no effect in the group of people with a potential risk, due to pre-existing health problems, may be because of the dilution of the true effect due to recall bias. CONCLUSIONS: Annoyance and disturbance due to road traffic noise is associated with a higher incidence of IHD. Prevalence of disease can be an important effect modifier of the relation between noise annoyance and health outcomes.

Aged↗
PubMed2001

Increased catecholamine levels in urine in subjects exposed to road traffic noise: the role of stress hormones in noise research.

The nocturnal excretion of catecholamines in urine was studied in 30-45-year-old women whose bedroom and/or living room were facing streets of varying traffic volume. The traffic volume of the streets was used as an indicator of noise exposure; adrenaline and noradrenaline concentrations were assessed as indicators of the outcome of the physiological stress. Significant associations between traffic volume and noradrenaline concentrations in urine were found with regard to the exposure of the bedroom (not the living room), indicating a higher chronic physiological arousal in noise-exposed subjects as compared to less exposed. Subjective measures of disturbance due to traffic noise were positively correlated with the noradrenaline level. However, this was only found in subjects where closing the window could not reduce the perceived disturbance, which points to the effectiveness of individual coping mechanisms. Stress hormones are useful indicators to study associations, mechanisms, and interactions between noise, health outcomes, and effect modifiers in epidemiological noise research.

Adaptation, Psychological↗
PubMed1999

[Hearing loss caused by leisure noise].

Although noise in general can induce hearing loss, environmental noise represents an important risk for children, teenagers and young adults. Epidemiological investigations now support the occurrence of an increasing number of irreversible hearing losses in these groups. Major causes of hearing loss are toys (guns), explosives and electroacoustically amplified music delivered by head sets or heard in discotheques and open air concerts. Clinical indications are discussed.

Adolescent↗
PubMed1997

Subjective work noise: a major risk factor in myocardial infarction.

The relationship between subjective work noise exposure and the risk of myocardial infarction (MI) was assessed in a population based case-control study. 395 MI patients (31-65 years) were compared to 2148 controls from a random population sample with the same agelsex distribution. The relative risk (RR) for MI-adjusted for control variables (smoking, age, social status, etc.)-was found to increase significantly and steadily with noise category. Subjective work noise exposure was the second greatest risk factor for MI after smoking. Possible bias due to overreporting of subjective noise exposure is discussed. Interdisciplinary studies on the relationship between cardiovascular diseases and work-related stressors including subjective and objective noise assessment are needed to quantify the risk of MI due to work noise.

Adult↗
PubMed1995

[Empirical studies of music listening habits of adolescents. Optimizing sound threshold limits for cassette players and discoteques].

The music habits of 569 pre-teens and teenagers, between 10 and 17 years of age, were examined and exposure to music from portable music players was compared to that in discotheques. Using a questionnaire we determined the length of time spent listening to portable music players and the frequency of disco visits. The individual level of the music heard through portable music players was measured as a free-field corrected short term mean level. The total music exposure (related to 40 hours per week) was calculated from the data and different assumed disco sound levels. We estimated the risk of ear damage according to ISO 1999 standards on the basis of the total music exposure, whereby the mean disco level was varied as a parameter in the range of 95 to 110 dB(A). Taking only the portable music players into account, one can expect that even after 5 years of music, approximately 5% of the total group would have a hearing loss of 20 dB. This percentage clearly rises when additional exposure is given in discotheques having music levels above 100 dB(A). We suggest therefore that for safety reasons the following sound levels should be observed: 90 dB(A) for portable music players and 95 dB(A) for discos. Through adherence to these levels we would expect that 1% of the young people in our study would have a hearing loss > 10 dB, whereas momentary exposure would result in a hearing loss > 10 dB of 10-20%. Therefore we believe that sound level limitations should be enacted immediately for portable music players and in discotheques.

Adolescent↗
PubMed1994

[Risk of hearing loss caused by listening to music with head phones].

Sound levels of music played from mini-cassette players via headphones were measured in a nonrepresentative group of 681 pupils whose ages were between 10 to 19 years. Each pupil completed a questionnaire giving the total time spent listening to music each day. The pupils set the music levels (free field corrected short time Leq) between 60 dB(A) and 110 dB(A). In the age group from 12-16 years, 10% chose 110 dB(A). Nearly 50% of the total group usually listened to music less than one hour per day, and just less than 10% listened for four or more hours. The energy equivalent of continuous sound pressure level for an exposure time of 8 h per day was for 55% Leq 8 h < 66 dB(A) and for 7% Leq 8 h > 95 dB(A) while 4% had an Leq 8 h > 105 dB(A). Estimation of the expected hearing losses (HL) were based on ISO 1999: about 10% of the total group were predicted to have a HL > 10 dB at 4 kHz after 5 years. After listening to sound in this manner 0.3% were expected to develop hearing losses at age 25 years that would be severe enough to substantially impair speech intelligibility.

Adolescent↗
PubMed1993

Road traffic noise, noise sensitivity and psychological disorder.

The relationship between traffic noise exposure and psychological morbidity was assessed using the population-based Caerphilly Collaborative Survey of 2398 men from Caerphilly, South Wales. The findings showed that traffic noise exposure levels were strongly associated with annoyance to noise. Noise-sensitive men were more likely to be highly annoyed by noise exposure than less noise-sensitive men. There was no direct association between noise exposure level and psychological morbidity but there were provocative interactions with noise sensitivity. The role of noise sensitivity is discussed as an indicator of vulnerability to environmental stressors and a measure of negative affectivity and over-reporting.

Cohort Studies↗
PubMed1993

Traffic noise as a risk factor for myocardial infarction.

The hypothesis that road traffic noise causes ischaemic heart disease (IHD) was tested in several epidemiological studies in middle-aged men. The studies carried out in Caerphilly (Wales) and Bristol-Speedwell (England) suggested the daytime (6-22 h) outdoor traffic noise level category 65-70 dB(A) as a threshold for noise effects detectable. Based on risk factors, a relative risk greater than 1 was calculated for the incidence of major IHD (RR = 1.1) in both samples for the subjects in this highest noise category, those in the quietest (51-56 dB(A)) serving as a reference using multiple models. Also the prevalence of myocardial infarction (RR = 1.2 and 1.1), ECG ischaemia (RR = 1.2 and 1.4) and any IHD disease (RR = 1.2 and 1.2) showed relative risks slightly greater than 1 in this noise group. In contradiction, the observed incidence of major IHD showed relative risks below 1 in both samples in this traffic noise group. The case-control studies carried out in Berlin revealed relative risks above 1 on the incidence (RR = 1.3 and 1.2) and the prevalence (RR = 1.2) of myocardial infarction at outdoor traffic noise levels in the range of 71-80 dB(A) serving the men in the category below 61 dB(A) as a reference. None of the findings was statistically significant. The relevance of the results is discussed.

Adult↗
PubMed1993

Road traffic noise, noise sensitivity and psychological disorder.

The relationship between traffic noise exposure and psychological morbidity was assessed using the population-based Caerphilly Collaborative Survey of 2398 men from Caerphilly, South Wales, UK. The findings showed that annoyance to noise was strongly associated with traffic noise exposure levels. Men with noise sensitivity were more likely to be highly annoyed by noise exposure than men with less noise sensitivity. There was no direct association between noise exposure level and psychological morbidity until the results were stratified by noise sensitivity. This revealed a gradient of increasing psychological morbidity with increasing noise level in the lower two tertiles of noise sensitivity. No gradient was found in the highest tertile of noise sensitivity. The role of noise sensitivity is argued to be either an indicator of vulnerability to environmental stressors or a measure of overreporting.

Anxiety Disorders↗
PubMed1992

[Mercury concentration in blood and urine--before and after the placement of dental amalgam fillings].

70 patients of dentist's surgery were given MOD amalgam fillings (non-gamma-2 amalgam) for molars. They were allocated for comparison to four groups defined by their treatment, i.e. the number of old and new restorations and whether a rubber dam was used. Blood and urine samples were collected at regular intervals before and during a 14-day period after treatment and tested for mercury concentration (Hg). Over the observation period the groups with the highest exposure (1-2 old restorations replaced by new ones) showed a tendency, in contrast to the less exposed groups (1 new filling with or without dam), towards increases (p less than 0.10) in group average Hg values of approx. 0.2 microgram/L (blood) and 0.3 microgram/g creatinine (urine) as acute treatment effects. The highest values recorded before and after the treatment, 3.3 micrograms/L (blood) and 16.5 micrograms/L (urine) are higher than normal but do not indicate any increase in the risk to health especially if they are not persistent.

Creatinine↗
PubMed1989

[The effect of music in discothèques on hearing ability].

The risk of hearing loss due to exposure to music in discotheques is discussed on the basis of VDI guideline No 2058, Bl 2. The hypothesis of a relationship was investigated in two empirical studies. In 204 pupils aged 13-19 years, a positive statistical relation of the 4 kHz hearing threshold on the music-hearing habits reported was observed. The difference between the extreme groups of noise exposure was approximately 4 dB (p less than 0.05) on a group average. The evaluation of audiograms of 3133 youths aged 16-20 years applying for a job performed during the medical tests required for employment, showed that those young people with a striking internal ear hearing loss of greater than 30 dB in the area of the C5 depression had spent significantly more time in discotheques than those without striking findings.

Adolescent↗